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Focal Segmental Glomerulosclerosis .

作者信息

Lassén Emelie, Daehn Ilse S

机构信息

Division of Nephrology, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York City, New York, USA.

出版信息

Kidney Int Rep. 2021 May 20;6(7):1758-1760. doi: 10.1016/j.ekir.2021.05.013. eCollection 2021 Jul.

DOI:10.1016/j.ekir.2021.05.013
PMID:34307972
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8258585/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c20e/8258585/ec156e18b944/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c20e/8258585/ec156e18b944/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c20e/8258585/ec156e18b944/gr1.jpg

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Focal Segmental Glomerulosclerosis .局灶节段性肾小球硬化
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Endothelial mitochondrial oxidative stress determines podocyte depletion in segmental glomerulosclerosis.内皮细胞线粒体氧化应激决定节段性肾小球硬化症中足细胞的耗竭。
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Renal Glomerular Expression of WT-1, TGF-β, VEGF, and ET-1 Immunostains in Murine Models of Focal and Segmental Glomerulosclerosis.局灶节段性肾小球硬化小鼠模型中WT-1、转化生长因子-β、血管内皮生长因子和内皮素-1免疫染色的肾小球表达
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Ultrastruct Pathol. 2018 Sep-Oct;42(5):430-439. doi: 10.1080/01913123.2018.1501125. Epub 2018 Oct 4.
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Endothelin receptor-A mediates degradation of the glomerular endothelial surface layer via pathologic crosstalk between activated podocytes and glomerular endothelial cells.内皮素受体-A 通过激活的足细胞和肾小球内皮细胞之间的病理串扰介导肾小球内皮表面层的降解。
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Role of podocytes for reversal of glomerulosclerosis and proteinuria in the aging kidney after endothelin inhibition.内皮素抑制后足细胞在衰老肾脏肾小球硬化和蛋白尿逆转中的作用。
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本文引用的文献

1
Endothelial Endothelin Receptor A Expression Is Associated With Podocyte Injury and Oxidative Stress in Patients With Focal Segmental Glomerulosclerosis.内皮素A受体在内皮细胞中的表达与局灶节段性肾小球硬化患者的足细胞损伤及氧化应激相关。
Kidney Int Rep. 2021 Apr 22;6(7):1939-1948. doi: 10.1016/j.ekir.2021.04.013. eCollection 2021 Jul.
2
New insights from SONAR indicate adding sodium glucose co-transporter 2 inhibitors to an endothelin receptor antagonist mitigates fluid retention and enhances albuminuria reduction.SONAR的新见解表明，在内皮素受体拮抗剂中添加钠-葡萄糖协同转运蛋白2抑制剂可减轻液体潴留并增强蛋白尿减少效果。
Kidney Int. 2021 Feb;99(2):346-349. doi: 10.1016/j.kint.2020.09.026. Epub 2020 Nov 2.
3
Endothelin receptor-A mediates degradation of the glomerular endothelial surface layer via pathologic crosstalk between activated podocytes and glomerular endothelial cells.
内皮素受体-A 通过激活的足细胞和肾小球内皮细胞之间的病理串扰介导肾小球内皮表面层的降解。
Kidney Int. 2019 Oct;96(4):957-970. doi: 10.1016/j.kint.2019.05.007. Epub 2019 May 22.
4
Urinary Markers of Oxidative Stress Are Associated With Albuminuria But Not GFR Decline.氧化应激的尿液标志物与白蛋白尿相关，但与肾小球滤过率下降无关。
Kidney Int Rep. 2017 Dec 7;3(3):573-582. doi: 10.1016/j.ekir.2017.11.020. eCollection 2018 May.
5
Endothelin-1 Induces Proteinuria by Heparanase-Mediated Disruption of the Glomerular Glycocalyx.内皮素-1通过肝素酶介导的肾小球糖萼破坏诱导蛋白尿。
J Am Soc Nephrol. 2016 Dec;27(12):3545-3551. doi: 10.1681/ASN.2015091070. Epub 2016 Mar 29.
6
Endothelial mitochondrial oxidative stress determines podocyte depletion in segmental glomerulosclerosis.内皮细胞线粒体氧化应激决定节段性肾小球硬化症中足细胞的耗竭。
J Clin Invest. 2014 Apr;124(4):1608-21. doi: 10.1172/JCI71195. Epub 2014 Mar 3.
7
Glomerular endothelial cell injury and damage precedes that of podocytes in adriamycin-induced nephropathy.阿霉素肾病中肾小球内皮细胞的损伤先于足细胞。
PLoS One. 2013;8(1):e55027. doi: 10.1371/journal.pone.0055027. Epub 2013 Jan 24.
8
Oxidative stress in primary glomerular diseases: a comparative study.原发性肾小球疾病中的氧化应激：一项对比研究。
Mol Cell Biochem. 2008 Apr;311(1-2):105-10. doi: 10.1007/s11010-008-9701-0. Epub 2008 Jan 25.
9
Molecular characterization of endothelin receptors.内皮素受体的分子特征
Trends Pharmacol Sci. 1992 Mar;13(3):103-8. doi: 10.1016/0165-6147(92)90038-8.