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TARG1 可防止毒性 DNA ADP-核糖基化。

TARG1 protects against toxic DNA ADP-ribosylation.

机构信息

Sir William Dunn School of Pathology, University of Oxford, Oxford OX1 3RE, UK.

Institute of Molecular Cancer Research, University of Zurich, 8057 Zurich, Switzerland.

出版信息

Nucleic Acids Res. 2021 Oct 11;49(18):10477-10492. doi: 10.1093/nar/gkab771.

Abstract

ADP-ribosylation is a modification that targets a variety of macromolecules and regulates a diverse array of important cellular processes. ADP-ribosylation is catalysed by ADP-ribosyltransferases and reversed by ADP-ribosylhydrolases. Recently, an ADP-ribosyltransferase toxin termed 'DarT' from bacteria, which is distantly related to human PARPs, was shown to modify thymidine in single-stranded DNA in a sequence specific manner. The antitoxin of DarT is the macrodomain containing ADP-ribosylhydrolase DarG, which shares striking structural homology with the human ADP-ribosylhydrolase TARG1. Here, we show that TARG1, like DarG, can reverse thymidine-linked DNA ADP-ribosylation. We find that TARG1-deficient human cells are extremely sensitive to DNA ADP-ribosylation. Furthermore, we also demonstrate the first detection of reversible ADP-ribosylation on genomic DNA in vivo from human cells. Collectively, our results elucidate the impact of DNA ADP-ribosylation in human cells and provides a molecular toolkit for future studies into this largely unknown facet of ADP-ribosylation.

摘要

ADP-核糖基化是一种针对多种生物大分子的修饰方式,可调节多种重要的细胞过程。ADP-核糖基化由 ADP-核糖基转移酶催化,由 ADP-核糖基水解酶逆转。最近,从细菌中分离出一种名为“DarT”的 ADP-核糖基转移酶毒素,它与人类 PARPs 有一定的亲缘关系,被证明能够以序列特异性的方式修饰单链 DNA 中的胸苷。DarT 的抗毒素是含有 ADP-核糖基水解酶结构域的 DarG,它与人类 ADP-核糖基水解酶 TARG1 具有惊人的结构同源性。在这里,我们表明 TARG1 与 DarG 一样,可以逆转与胸苷相连的 DNA ADP-核糖基化。我们发现 TARG1 缺陷的人类细胞对 DNA ADP-核糖基化极其敏感。此外,我们还首次在体内从人类细胞中检测到基因组 DNA 上可逆的 ADP-核糖基化。总之,我们的研究结果阐明了 DNA ADP-核糖基化在人类细胞中的作用,并为未来对这一未知 ADP-核糖基化领域的研究提供了分子工具。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ed5b/8501950/b8e2ef809748/gkab771fig1.jpg

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