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感染、天体影响与婴儿猝死综合征:一种新范式。

Infection, Celestial Influences, and Sudden Infant Death Syndrome: A New Paradigm.

作者信息

Goldwater Paul N, Oberg Edward O

机构信息

Pathology-Infectious Diseases and Clinical Microbiology, Adelaide Medical School, University of Adelaide, Adelaide, AUS.

Mechanical Engineering, University of Minnesota, Minnesota, USA.

出版信息

Cureus. 2021 Aug 26;13(8):e17449. doi: 10.7759/cureus.17449. eCollection 2021 Aug.

DOI:10.7759/cureus.17449
PMID:34589355
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8463918/
Abstract

The etiology of sudden infant death syndrome (SIDS) still remains unclear. This situation would seem unprecedented for 21st-century medical science. This article explores scientific fields that have not been largely considered in investigating the etiology of SIDS so far. In this study, we examined previously ignored studies on heliobiology, celestial influences, and SIDS in the non-medical literature in an attempt to answer the following questions: is there a relationship between sunspot/solar activity and the occurrence of SIDS? Could there be alternative reasons for the decline in SIDS incidences in the 1990s that were originally attributed to the "Back-to-Sleep" campaign? We note that the decline coincided with the ~11-year cyclical diminution in sunspot numbers (SSNs). The SSN/SIDS relationship does not necessarily imply causality; however, it supports published data regarding sunspots, Schumann resonance, and geomagnetic effects. How solar energy could adversely influence a baby's existence remains conjectural. Observations in this respect suggest pathways involving melatonin and/or infection/inflammation.

摘要

婴儿猝死综合征(SIDS)的病因仍不明确。对于21世纪的医学科学而言,这种情况似乎前所未有的。本文探讨了迄今为止在调查SIDS病因时未被充分考虑的科学领域。在本研究中,我们查阅了非医学文献中先前被忽视的关于日地生物学、天体影响和SIDS的研究,试图回答以下问题:太阳黑子/太阳活动与SIDS的发生之间是否存在关联?20世纪90年代SIDS发病率下降,最初归因于“仰睡”运动,是否可能存在其他原因?我们注意到,这种下降与太阳黑子数(SSNs)约11年的周期性减少相吻合。SSN与SIDS之间的关系不一定意味着因果关系;然而,它支持了关于太阳黑子、舒曼共振和地磁效应的已发表数据。太阳能如何对婴儿的生存产生不利影响仍属推测。这方面的观察结果提示了涉及褪黑素和/或感染/炎症的途径。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26d4/8463918/7afc3f2510ba/cureus-0013-00000017449-i01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26d4/8463918/7afc3f2510ba/cureus-0013-00000017449-i01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26d4/8463918/7afc3f2510ba/cureus-0013-00000017449-i01.jpg

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Increased thymus weight in sudden infant death syndrome compared to controls: The role of sub-clinical infections.与对照组相比,婴儿猝死综合征患者的胸腺重量增加:亚临床感染的作用。
Am J Hum Biol. 2021 Nov;33(6):e23528. doi: 10.1002/ajhb.23528. Epub 2020 Oct 26.
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Violet-light suppression of thermogenesis by opsin 5 hypothalamic neurons.视蛋白 5 下丘脑神经元对产热的紫光抑制。
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SIDS, prone sleep position and infection: An overlooked epidemiological link in current SIDS research? Key evidence for the "Infection Hypothesis".
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On the correlation between solar activity and large earthquakes worldwide.关于太阳活动与全球大地震之间的相关性
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