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专刊编辑寄语:衰老的分子基础

Editorial for the Special Issue "Molecular Bases of Senescence".

机构信息

IIM-Interuniversity Institute of Myology, University "G. d'Annunzio" of Chieti-Pescara, 66100 Chieti, Italy.

Campus of Free University of Alcatraz, Santa Cristina di Gubbio, 06100 Perugia, Italy.

出版信息

Int J Mol Sci. 2021 Nov 2;22(21):11873. doi: 10.3390/ijms222111873.

DOI:10.3390/ijms222111873
PMID:34769304
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8585045/
Abstract

The increasing life expectancy of populations worldwide represents the most evident success of the last century thanks to varying interacting social and medical achievements [...].

摘要

全球人口的预期寿命不断延长,这是上个世纪最显著的成功,这要归功于各种相互作用的社会和医学成就[...]。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/33a2/8585045/d822da1e6d21/ijms-22-11873-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/33a2/8585045/e5c45ded58dd/ijms-22-11873-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/33a2/8585045/b415947afb85/ijms-22-11873-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/33a2/8585045/d822da1e6d21/ijms-22-11873-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/33a2/8585045/e5c45ded58dd/ijms-22-11873-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/33a2/8585045/b415947afb85/ijms-22-11873-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/33a2/8585045/d822da1e6d21/ijms-22-11873-g003.jpg

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本文引用的文献

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Biological Aspects of Selected Myokines in Skeletal Muscle: Focus on Aging.某些肌肉因子的生物学特性在骨骼肌中的表现:聚焦于衰老。
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2
Chronological Age Affects MSC Senescence In Vitro-A Systematic Review.增龄对间充质干细胞体外衰老的影响:系统评价。
Int J Mol Sci. 2021 Jul 26;22(15):7945. doi: 10.3390/ijms22157945.
3
Distinct Age-Specific miRegulome Profiling of Isolated Small and Large Intestinal Epithelial Cells in Mice.小鼠分离的小肠和大肠上皮细胞的特定年龄特异性的 miRegulome 特征分析。
Int J Mol Sci. 2021 Mar 29;22(7):3544. doi: 10.3390/ijms22073544.
4
Understanding the Potential Role of Sirtuin 2 on Aging: Consequences of SIRT2.3 Overexpression in Senescence.理解 Sirtuin 2 在衰老中的潜在作用:SIRT2.3 过表达在衰老中的后果。
Int J Mol Sci. 2021 Mar 18;22(6):3107. doi: 10.3390/ijms22063107.
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Aged Skeletal Muscle Retains the Ability to Remodel Extracellular Matrix for Degradation of Collagen Deposition after Muscle Injury.衰老骨骼肌在肌肉损伤后仍能重塑细胞外基质以降解胶原蛋白沉积。
Int J Mol Sci. 2021 Feb 20;22(4):2123. doi: 10.3390/ijms22042123.
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Non-Coding RNAs in the Transcriptional Network That Differentiates Skeletal Muscles of Sedentary from Long-Term Endurance- and Resistance-Trained Elderly.非编码 RNA 在转录网络中的作用,区分了久坐的老年人与长期耐力和抗阻训练的老年人的骨骼肌。
Int J Mol Sci. 2021 Feb 3;22(4):1539. doi: 10.3390/ijms22041539.
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CK2 Down-Regulation Increases the Expression of Senescence-Associated Secretory Phenotype Factors through NF-κB Activation.CK2 下调通过 NF-κB 激活增加衰老相关分泌表型因子的表达。
Int J Mol Sci. 2021 Jan 2;22(1):406. doi: 10.3390/ijms22010406.
8
Linking ABCC6 Deficiency in Primary Human Dermal Fibroblasts of PXE Patients to p21-Mediated Premature Cellular Senescence and the Development of a Proinflammatory Secretory Phenotype.将原发性进行性皮肤弹力纤维溶解症(PXE)患者的原代表皮成纤维细胞中的ABCC6缺陷与p21介导的细胞早衰及促炎分泌表型的发展联系起来。
Int J Mol Sci. 2020 Dec 18;21(24):9665. doi: 10.3390/ijms21249665.
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Int J Mol Sci. 2020 Nov 30;21(23):9130. doi: 10.3390/ijms21239130.
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Int J Mol Sci. 2020 Sep 15;21(18):6766. doi: 10.3390/ijms21186766.