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在国家篮球协会球员中,向篮筐进攻的倾向与前交叉韧带撕裂风险增加相关:一项队列研究。

Tendency of Driving to the Basket Is Associated With Increased Risk of Anterior Cruciate Ligament Tears in National Basketball Association Players: A Cohort Study.

作者信息

Schultz Blake J, Thomas Kevin A, Cinque Mark, Harris Joshua D, Maloney William J, Abrams Geoffrey D

机构信息

Department of Orthopaedics, Stanford University, Redwood City, California, USA.

Houston Methodist Orthopedics & Sports Medicine, Houston, Texas, USA.

出版信息

Orthop J Sports Med. 2021 Nov 5;9(11):23259671211052953. doi: 10.1177/23259671211052953. eCollection 2021 Nov.

DOI:10.1177/23259671211052953
PMID:34778484
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8573492/
Abstract

BACKGROUND

Driving to the basket in basketball involves acceleration, deceleration, and lateral movements, which may expose players to increased anterior cruciate ligament (ACL) injury risk. It is unknown whether players who heavily rely on driving have decreased performance on returning to play after ACL reconstruction (ACLR).

HYPOTHESIS

Players with a greater tendency to drive to the basket would be more likely to tear their ACL versus noninjured controls and would experience decreased performance when returning to play after ACLR.

STUDY DESIGN

Case-control study; Level of evidence, 3.

METHODS

Season-level performance statistics and ACL injuries were aggregated for National Basketball Association (NBA) seasons between 1980 and 2017 from publicly available sources. Players' tendency to drive was calculated using 49 common season-level performance metrics. Each ACL-injured player was matched with 2 noninjured control players by age, league experience, and style of play metrics. Points, playing minutes, driving, and 3-point shooting tendencies were compared between players with ACL injuries and matched controls. Independent-samples test was utilized for comparisons.

RESULTS

Of 86 players with a total of 96 ACL tears identified in the NBA, 50 players were included in the final analysis. Players who experienced an ACL tear had a higher career-average drive tendency than controls ( = .047). Players with career-average drive tendency ≥1 standard deviation above the mean were more likely to tear their ACL than players with drive tendency <1 standard deviation (5.2% vs 2.7%; = .026). There was no significant difference in total postinjury career points ( = .164) or career minutes ( = .237) between cases and controls. There was also no significant change in drive tendency ( = .152) or 3-point shooting tendency ( = .508) after return to sport compared with controls.

CONCLUSION

NBA players with increased drive tendency were more likely to tear their ACL. However, players who were able to return after ACLR did not underperform compared with controls and did not alter their style of play compared with the normal changes seen with age. This information can be used to target players with certain playing styles for ACL injury prevention programs.

摘要

背景

篮球比赛中向篮筐突破涉及加速、减速和横向移动,这可能会增加球员前交叉韧带(ACL)损伤的风险。目前尚不清楚严重依赖突破的球员在ACL重建(ACLR)后重返赛场时表现是否会下降。

假设

与未受伤的对照组相比,更倾向于向篮筐突破的球员更有可能撕裂其ACL,并且在ACLR后重返赛场时表现会下降。

研究设计

病例对照研究;证据等级,3级。

方法

从公开资料中汇总了1980年至2017年美国职业篮球联赛(NBA)赛季的赛季水平表现统计数据和ACL损伤情况。使用49个常见的赛季水平表现指标计算球员的突破倾向。每位ACL损伤的球员与2名未受伤的对照球员按年龄、联赛经验和比赛风格指标进行匹配。比较ACL损伤球员与匹配对照组之间的得分、上场时间、突破和三分球投篮倾向。采用独立样本t检验进行比较。

结果

在NBA中确定的86名共有96次ACL撕裂的球员中,50名球员纳入最终分析。经历ACL撕裂的球员职业生涯平均突破倾向高于对照组(P = 0.047)。职业生涯平均突破倾向≥高于均值1个标准差的球员比突破倾向<1个标准差的球员更有可能撕裂其ACL(5.2%对2.7%;P = 0.026)。病例组和对照组之间伤后职业生涯总得分(P = 0.164)或职业生涯上场时间(P = 0.237)没有显著差异。与对照组相比,重返赛场后突破倾向(P = 0.152)或三分球投篮倾向(P = 0.508)也没有显著变化。

结论

突破倾向增加的NBA球员更有可能撕裂其ACL。然而,ACLR后能够重返赛场的球员与对照组相比表现并不逊色,并且与随年龄增长出现的正常变化相比,他们的比赛风格没有改变。这些信息可用于针对具有特定比赛风格的球员开展ACL损伤预防计划。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/aba5708b57b6/10.1177_23259671211052953-fig5.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/8aca2201d5ab/10.1177_23259671211052953-fig1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/e7cb23a2e685/10.1177_23259671211052953-fig2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/0f2e1a8d0818/10.1177_23259671211052953-fig3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/42f72c4f8d06/10.1177_23259671211052953-fig4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/aba5708b57b6/10.1177_23259671211052953-fig5.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/8aca2201d5ab/10.1177_23259671211052953-fig1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/e7cb23a2e685/10.1177_23259671211052953-fig2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/0f2e1a8d0818/10.1177_23259671211052953-fig3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/42f72c4f8d06/10.1177_23259671211052953-fig4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/118e/8573492/aba5708b57b6/10.1177_23259671211052953-fig5.jpg

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