勘误：高血压相关肠道微生物群的差异分析。
Erratum: Differential Analysis of Hypertension-Associated Intestinal Microbiota.
作者信息
Dan Xie, Mushi Zhang, Baili Wang, Han Lin, Enqi Wu, Huanhu Zhao, Shuchun Li
机构信息
School of Pharmacy, Minzu University of China, Key Laboratory of Ethnomedicine (Minzu University of China), Ministry of education, Beijing 100081, P.R. China.
School Hospital, Minzu University of China, Beijing 100081, China.
出版信息
Int J Med Sci. 2021 Oct 8;18(16):3748. doi: 10.7150/ijms.65972. eCollection 2021.
Abstract
[This corrects the article DOI: 10.7150/ijms.29322.].
摘要
[本文更正了文章的数字对象标识符：10.7150/ijms.29322。]
相似文献
1
Erratum: Differential Analysis of Hypertension-Associated Intestinal Microbiota.勘误：高血压相关肠道微生物群的差异分析。
Int J Med Sci. 2021 Oct 8;18(16):3748. doi: 10.7150/ijms.65972. eCollection 2021.
2
Erratum: Role of Sirolimus in renal tubular apoptosis in response to unilateral ureteral obstruction: Erratum.勘误：西罗莫司在单侧输尿管梗阻所致肾小管凋亡中的作用：勘误
Int J Med Sci. 2022 Mar 3;19(3):488-489. doi: 10.7150/ijms.71282. eCollection 2022.
3
Erratum: PDLSCs Regulate Angiogenesis of Periodontal Ligaments via VEGF Transferred by Exosomes in Periodontitis: Erratum.勘误：牙周炎中牙周膜干细胞通过外泌体转运的VEGF调节牙周膜血管生成：勘误
Int J Med Sci. 2022 May 3;19(5):833. doi: 10.7150/ijms.74583. eCollection 2022.
4
Erratum: LncRNA AFAP1-AS Functions as a Competing Endogenous RNA to Regulate RAP1B Expression by sponging miR-181a in the HSCR: Erratum.勘误：长链非编码RNA AFAP1-AS作为竞争性内源性RNA，通过在先天性巨结肠症中吸附miR-181a来调控RAP1B表达：勘误。
Int J Med Sci. 2018 Oct 2;15(13):1472. doi: 10.7150/ijms.29742. eCollection 2018.
5
Erratum: Estrogen receptor α/prolactin receptor bilateral crosstalk promotes bromocriptine resistance in prolactinomas: Erratum.勘误：雌激素受体α/催乳素受体双侧串扰促进催乳素瘤对溴隐亭的耐药性：勘误
Int J Med Sci. 2022 May 3;19(5):831-832. doi: 10.7150/ijms.71659. eCollection 2022.
6
Erratum: Association of Genes Variants in RANKL/RANK/OPG Signaling Pathway with the Development of Osteonecrosis of the Femoral Head in Chinese Population: Erratum.勘误：RANKL/RANK/OPG信号通路基因变异与中国人群股骨头坏死发生的关联：勘误
Int J Med Sci. 2018 Aug 10;15(12):1355. doi: 10.7150/ijms.29056. eCollection 2018.
7
Erratum: PRMT4 promotes hepatocellular carcinoma progression by activating AKT/mTOR signaling and indicates poor prognosis: Erratum.勘误：PRMT4通过激活AKT/mTOR信号通路促进肝细胞癌进展并提示预后不良：勘误。
Int J Med Sci. 2022 Mar 3;19(3):486-487. doi: 10.7150/ijms.71614. eCollection 2022.
8
Erratum: Medicine, Volume 95, Issue 23: Erratum.勘误：《医学》，第95卷，第23期：勘误。
Medicine (Baltimore). 2016 Jul 18;95(28):e0916. doi: 10.1097/01.md.0000489580.04709.16. eCollection 2016 Jul.
9
Erratum: Medicine, Volume 95, Issue 24: Erratum.勘误：《医学》，第95卷，第24期：勘误。
Medicine (Baltimore). 2016 Aug 7;95(31):e5074. doi: 10.1097/01.md.0000490009.39850.74. eCollection 2016 Aug.
10
Erratum: CD8 T cell survival in lethal fungal sepsis was ameliorated by T-cell-specific mTOR deletion: Erratum.勘误：T细胞特异性mTOR缺失改善了致死性真菌败血症中CD8 T细胞的存活：勘误。
Int J Med Sci. 2022 Mar 3;19(3):484-485. doi: 10.7150/ijms.71620. eCollection 2022.
引用本文的文献
1
Relationships among gut microbes, the interleukin family, and hypertension: a mediation Mendelian randomization study.肠道微生物、白细胞介素家族与高血压之间的关系：一项中介孟德尔随机化研究。
Front Nutr. 2023 Nov 28;10:1293170. doi: 10.3389/fnut.2023.1293170. eCollection 2023.
2
Emodin Inhibited Pathological Cardiac Hypertrophy in Response to Angiotensin-Induced Hypertension and Altered the Gut Microbiome.大黄素抑制血管紧张素诱导的高血压引起的病理性心肌肥厚，并改变肠道微生物组。
Biomolecules. 2023 Aug 22;13(9):1274. doi: 10.3390/biom13091274.
3
Causality of gut microbiome and hypertension: A bidirectional mendelian randomization study.肠道微生物群与高血压的因果关系：一项双向孟德尔随机化研究。
Front Cardiovasc Med. 2023 May 4;10:1167346. doi: 10.3389/fcvm.2023.1167346. eCollection 2023.
4
Ginger essential oil and citral ameliorates atherosclerosis in ApoE mice by modulating trimethylamine-N-oxide and gut microbiota.姜精油和柠檬醛通过调节氧化三甲胺和肠道微生物群改善载脂蛋白E基因敲除小鼠的动脉粥样硬化。
NPJ Sci Food. 2023 May 20;7(1):19. doi: 10.1038/s41538-023-00196-0.
本文引用的文献
1
Differential Analysis of Hypertension-Associated Intestinal Microbiota.高血压相关肠道微生物组的差异分析。
Int J Med Sci. 2019 Jun 2;16(6):872-881. doi: 10.7150/ijms.29322. eCollection 2019.
Zotero 插件