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严重内翻型踝骨关节炎的距下关节特征性骨形态变化。

Characteristic Bone Morphology Change of the Subtalar Joint in Severe Varus Ankle Osteoarthritis.

机构信息

Department of Orthopaedic Surgery, Graduate School of Biomedical Sciences, Hiroshima University, Minami-ku, Hiroshima, Japan.

Department of Orthopaedic Surgery, Graduate School of Biomedical Sciences, Hiroshima University, Minami-ku, Hiroshima, Japan; Medical Center for Translation and Clinical Research, Hiroshima University Hospital, Minami-ku, Hiroshima, Japan.

出版信息

J Foot Ankle Surg. 2022 May-Jun;61(3):627-632. doi: 10.1053/j.jfas.2021.10.026. Epub 2021 Oct 29.

Abstract

The subtalar joint has a possible compensatory mechanism for supramalleolar deformities; the failure of this mechanism causes the progression of ankle osteoarthritis. However, the reason for this failure has not been fully elucidated. This study aimed to evaluate the characteristics of the morphologic changes in the subtalar joint in varus ankle osteoarthritis using computed tomography. The study included 30 patients with severe osteoarthritis (modified Kellgren-Lawrence classification grade ≥ 3; mean age: 68.5 years) and 30 patients without- or with early osteoarthritis (grade 0-1; mean age: 43.0 years) as the control group. The location of cysts, osteophyte formation in the subtalar joint, and thickness of the subchondral bone plate were evaluated. In the osteoarthritis group, cyst formation was observed on the posterolateral side of the posterior facet of the calcaneus in 6 cases (20%) and of the talus in 7 cases (23.3%). Osteophyte formation was observed in the talus in 21 cases (70.0%) and in the calcaneus in 29 cases (96.6%). Osteophyte formation was observed on the posterior or lateral side of the posterior facet, and osteophyte contact between the talus and calcaneus was observed. The subchondral bone plate of the posterior medial side of the posterior facet of the talus was significantly thicker in the osteoarthritis group. The subtalar joint is less affected in severe varus ankle osteoarthritis containing a thickened subchondral bone plate in the posteromedial aspect of the posterior talar facet.

摘要

距下关节可能存在一种对踝上畸形的代偿机制;该机制的失败导致踝关节骨关节炎的进展。然而,其失败的原因尚未完全阐明。本研究旨在通过计算机断层扫描评估内翻型踝关节骨关节炎距下关节形态变化的特征。该研究包括 30 例严重骨关节炎患者(改良 Kellgren-Lawrence 分级≥3;平均年龄:68.5 岁)和 30 例无或早期骨关节炎患者(0-1 级;平均年龄:43.0 岁)作为对照组。评估了距下关节的囊肿位置、骨赘形成和软骨下骨板厚度。在骨关节炎组中,6 例(20%)后关节跟骨后外侧和 7 例(23.3%)距骨后外侧出现囊肿形成。21 例(70.0%)距骨和 29 例(96.6%)跟骨出现骨赘形成。骨赘形成位于后关节的后外侧或侧方,可见距骨和跟骨之间的骨赘接触。距骨后关节后内侧的软骨下骨板在骨关节炎组中明显增厚。在包含距骨后关节后内侧软骨下骨板增厚的严重内翻型踝关节骨关节炎中,距下关节受影响较小。

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