Thompson P D, Mastaglia F L, Carroll W M
J Neurol Neurosurg Psychiatry. 1986 Feb;49(2):128-35. doi: 10.1136/jnnp.49.2.128.
The clinical and visual evoked potential (VEP) findings were analysed in 18 patients with anterior ischaemic optic neuropathy. The VEP studies showed a variety of abnormalities which could be interpreted as being the result of subcomponent interaction consequent upon loss or attenuation of the normal macular-derived P100 component. Delay of normal VEP subcomponents was not seen. The VEP findings were non-specific but pointed to a severe disturbance of transmission in optic nerve fibres subserving central vision. No significant changes were observed with time in most cases indicating a static monophasic process with no significant recovery.
对18例前部缺血性视神经病变患者的临床及视觉诱发电位（VEP）结果进行了分析。VEP研究显示出多种异常，这些异常可解释为正常黄斑源性P100成分丧失或减弱后子成分相互作用的结果。未发现正常VEP子成分延迟。VEP结果不具有特异性，但提示服务于中心视力的视神经纤维传导存在严重障碍。大多数病例中未观察到随时间的显著变化，表明这是一个静态单相过程，无明显恢复。
