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灰口铸铁铸造厂中的矽肺病。一种古老疾病的持续存在。

Silicosis in a grey iron foundry. The persistence of an ancient disease.

作者信息

Landrigan P J, Cherniack M G, Lewis F A, Catlett L R, Hornung R W

出版信息

Scand J Work Environ Health. 1986 Feb;12(1):32-9. doi: 10.5271/sjweh.2181.

Abstract

An industrial hygiene and medical survey was conducted in an iron foundry to study the occurrence of silicosis. Breathing zone exposures to respirable crystalline silica had been very high in 1977 [1 045 micrograms/m3 (geometric mean) for coremakers and 198 micrograms/m3 for fettlers]; exposures in 1980 and 1982 were substantially lower. A radiographic evaluation of 188 workers revealed silicosis in 18 (9.6%). Eight had category 1 profusion of small rounded pulmonary lesions (by the 1980 classification of the International Labour Office); two had category 2; and eight had category 3. Two had progressive massive fibrosis. Four workers without silicosis in 1977 had developed lesions by 1980. The prevalence increased from 1.5% among workers employed less than 20 years to 53% among longer term workers. No association was found between the prevalence of silicosis and cigarette smoking. Chronic cough was more common in workers with heavy current dust exposure than in those with light exposure, more common in smokers than in nonsmokers, and more common in silicotics than in nonsilicotics. A multiplicative interaction existed between dust exposure and smoking in the etiology of cough. Silicosis continues to exist in American foundries. Cigarette smoking does not contribute to the causation of silicosis, but it aggravates the attendant respiratory symptoms.

摘要

在一家铸铁厂进行了工业卫生与医学调查,以研究矽肺病的发病情况。1977年,呼吸带可吸入结晶二氧化硅的暴露水平非常高(型芯工为1045微克/立方米(几何均值),清铲工为198微克/立方米);1980年和1982年的暴露水平大幅降低。对188名工人进行的X光评估显示,18人(9.6%)患有矽肺病。8人有1级小圆形肺病变(根据国际劳工组织1980年的分类);2人有2级;8人有3级。2人有进行性大块纤维化。1977年没有矽肺病的4名工人到1980年出现了病变。患病率从工作不到20年的工人中的1.5%上升到长期工人中的53%。未发现矽肺病患病率与吸烟之间存在关联。目前粉尘暴露量大的工人比暴露量小的工人慢性咳嗽更常见,吸烟者比不吸烟者更常见,矽肺病患者比非矽肺病患者更常见。在咳嗽的病因中,粉尘暴露与吸烟之间存在相乘交互作用。矽肺病在美国的铸造厂中仍然存在。吸烟不会导致矽肺病,但会加重随之而来的呼吸道症状。

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