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Spontaneous Cholemia in C57BL/6 Mice Predisposes to Liver Cancer in NASH.

作者信息

Gallage Suchira, Ali Adnan, Barragan Avila Jose Efren, Herebian Diran, Karimi Mohammad M, Irvine Elaine E, McHugh Domhnall, Schneider Anne T, Vucur Mihael, Keitel Verena, Gil Jesús, Withers Dominic J, Luedde Tom, Heikenwalder Mathias

机构信息

Division of Chronic Inflammation and Cancer, German Cancer Research Center (DKFZ), Heidelberg, Germany.

Department of General Pediatrics, Neonatology and Pediatric Cardiology, Medical Faculty, University Hospital Düsseldorf, Heinrich Heine University, Düsseldorf, Germany.

出版信息

Cell Mol Gastroenterol Hepatol. 2022;13(3):875-878. doi: 10.1016/j.jcmgh.2021.11.012. Epub 2021 Dec 6.

Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/05fd/8804272/a68bbea26393/gr1.jpg

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The therapeutic landscape of hepatocellular carcinoma.
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Liver Inflammation and Hepatobiliary Cancers.
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From NASH to HCC: current concepts and future challenges.
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Bile acids activate YAP to promote liver carcinogenesis.
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Diet-induced mouse model of fatty liver disease and nonalcoholic steatohepatitis reflecting clinical disease progression and methods of assessment.
Am J Physiol Gastrointest Liver Physiol. 2013 Oct 1;305(7):G483-95. doi: 10.1152/ajpgi.00079.2013. Epub 2013 Jul 25.
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Heterogeneous metabolic adaptation of C57BL/6J mice to high-fat diet.
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