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中毒性休克综合征毒素-1的生物学活性研究。I. 毒素刺激T细胞后的增殖反应及白细胞介素2的产生

Study of the biological activities of toxic shock syndrome toxin-1. I. Proliferative response and interleukin 2 production by T cells stimulated with the toxin.

作者信息

Uchiyama T, Kamagata Y, Wakai M, Yoshioka M, Fujikawa H, Igarashi H

出版信息

Microbiol Immunol. 1986;30(5):469-83. doi: 10.1111/j.1348-0421.1986.tb02973.x.

DOI:10.1111/j.1348-0421.1986.tb02973.x
PMID:3489157
Abstract

The mitogenic and interleukin 2 (IL 2) production-inducing effects of toxic shock syndrome toxin-1 (TSST-1) on murine lymphocytes were investigated. TSST-1, an exotoxin produced by Staphylococcus aureus recovered from patients with toxic shock syndrome (TSS), is thought to be a causative agent of the syndrome. TSST-1 was mitogenic for splenic T cells and peanut agglutinin (PNA)-negative thymocytes, but not for T cell-depleted spleen cells, PNA-positive thymocytes or IL 2-dependent CTLL 2-cells. A factor mitogenic for CTCC-2 cells with a molecular weight of 30-35 kdaltons was obtained by stimulating spleen cells with TSST-1 and it was absorbed by CTLL-2 cells, indicating that the factor is IL 2. For substantial amounts of IL 2 to be produced, 10 ng or more of TSST-1 per ml and 48 hr or more of incubation were required. Removal of T cells abrogated the IL 2 production by spleen cells. T cells obtained by the nylon wool column method alone produced IL 2 on TSST-1 stimulation in the presence of either macrophages or a macrophage lysate containing interleukin 1. However, T cells obtained by a combination of the nylon wool column method and anti-Ia antibody treatment produced IL 2 in the presence of macrophages but not of the macrophage lysate, indicating that IL 2 production by TSST-1-stimulated T cells is absolutely dependent on the presence of accessory cells.

摘要

研究了中毒性休克综合征毒素-1(TSST-1)对小鼠淋巴细胞的促有丝分裂作用以及诱导白细胞介素2(IL-2)产生的作用。TSST-1是从中毒性休克综合征(TSS)患者体内分离出的金黄色葡萄球菌产生的一种外毒素,被认为是该综合征的致病因子。TSST-1对脾T细胞和花生凝集素(PNA)阴性胸腺细胞有促有丝分裂作用,但对T细胞缺失的脾细胞、PNA阳性胸腺细胞或IL-2依赖的CTLL-2细胞无此作用。用TSST-1刺激脾细胞可获得一种对CTCC-2细胞有促有丝分裂作用、分子量为30 - 35千道尔顿的因子,且该因子可被CTLL-2细胞吸收,表明该因子为IL-2。要产生大量的IL-2,每毫升需要10纳克或更多的TSST-1,且培养时间需48小时或更长。去除T细胞可消除脾细胞产生IL-2的能力。仅通过尼龙毛柱法获得的T细胞在巨噬细胞或含有白细胞介素1的巨噬细胞裂解物存在的情况下,经TSST-1刺激可产生IL-2。然而,通过尼龙毛柱法和抗Ia抗体处理相结合获得的T细胞在巨噬细胞存在时可产生IL-2,但在巨噬细胞裂解物存在时则不能,这表明TSST-1刺激的T细胞产生IL-2绝对依赖于辅助细胞的存在。

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Study of the biological activities of toxic shock syndrome toxin-1. I. Proliferative response and interleukin 2 production by T cells stimulated with the toxin.中毒性休克综合征毒素-1的生物学活性研究。I. 毒素刺激T细胞后的增殖反应及白细胞介素2的产生
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