• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Correction to: Elevated microRNA-129-5p level ameliorates neuroinflammation and blood-spinal cord barrier damage after ischemia-reperfusion by inhibiting HMGB1 and the TLR3-cytokine pathway.

作者信息

Li Xiao-Qian, Chen Feng-Shou, Tan Wen-Fei, Fang Bo, Zhang Zai-Li, Ma Hong

机构信息

Department of Anesthesiology, First Affiliated Hospital, China Medical University, Shenyang, 110001, Liaoning, China.

出版信息

J Neuroinflammation. 2021 Dec 28;18(1):308. doi: 10.1186/s12974-021-02345-2.

DOI:10.1186/s12974-021-02345-2
PMID:34963474
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8715607/
Abstract
摘要

相似文献

1
Correction to: Elevated microRNA-129-5p level ameliorates neuroinflammation and blood-spinal cord barrier damage after ischemia-reperfusion by inhibiting HMGB1 and the TLR3-cytokine pathway.对《缺血再灌注后微小RNA-129-5p水平升高通过抑制高迁移率族蛋白B1及Toll样受体3-细胞因子途径改善神经炎症和血脊髓屏障损伤》一文的更正
J Neuroinflammation. 2021 Dec 28;18(1):308. doi: 10.1186/s12974-021-02345-2.
2
Elevated microRNA-129-5p level ameliorates neuroinflammation and blood-spinal cord barrier damage after ischemia-reperfusion by inhibiting HMGB1 and the TLR3-cytokine pathway.高表达 microRNA-129-5p 通过抑制 HMGB1 和 TLR3-细胞因子通路减轻缺血再灌注后神经炎症和血脊髓屏障损伤。
J Neuroinflammation. 2017 Oct 23;14(1):205. doi: 10.1186/s12974-017-0977-4.
3
Dexmedetomidine Preconditioning Ameliorates Inflammation and Blood-Spinal Cord Barrier Damage After Spinal Cord Ischemia-Reperfusion Injury by Down-Regulation High Mobility Group Box 1-Toll-Like Receptor 4-Nuclear Factor κB Signaling Pathway.右美托咪定预处理通过下调高迁移率族蛋白 B1- Toll 样受体 4-核因子 κB 信号通路减轻脊髓缺血再灌注损伤后的炎症和血脊髓屏障损伤。
Spine (Phila Pa 1976). 2019 Jan 15;44(2):E74-E81. doi: 10.1097/BRS.0000000000002772.
4
Altered expression of MiR-186-5p and its target genes after spinal cord ischemia-reperfusion injury in rats.大鼠脊髓缺血再灌注损伤后 miR-186-5p 及其靶基因表达的改变。
Neurosci Lett. 2020 Jan 23;718:134669. doi: 10.1016/j.neulet.2019.134669. Epub 2019 Dec 2.
5
MiR-27a ameliorates inflammatory damage to the blood-spinal cord barrier after spinal cord ischemia: reperfusion injury in rats by downregulating TICAM-2 of the TLR4 signaling pathway.微小RNA-27a通过下调Toll样受体4信号通路的TICAM-2改善脊髓缺血再灌注损伤后血脊髓屏障的炎性损伤：大鼠实验研究
J Neuroinflammation. 2015 Feb 7;12:25. doi: 10.1186/s12974-015-0246-3.
6
Downregulation of Long Noncoding RNA TUG1 Attenuates MTDH-Mediated Inflammatory Damage via Targeting miR-29b-1-5p After Spinal Cord Ischemia Reperfusion.长链非编码 RNA TUG1 下调通过靶向 miR-29b-1-5p 减轻脊髓缺血再灌注后 MTDH 介导的炎症损伤。
J Neuropathol Exp Neurol. 2021 Feb 22;80(3):254-264. doi: 10.1093/jnen/nlaa138.
7
miR-129-5p ameliorates ischemia-reperfusion injury by targeting HMGB1 in myocardium.微小RNA-129-5p通过靶向心肌中的高迁移率族蛋白B1改善缺血再灌注损伤。
Gen Physiol Biophys. 2020 Sep;39(5):461-470. doi: 10.4149/gpb_2020021.
8
Key genes expressed in different stages of spinal cord ischemia/reperfusion injury.在脊髓缺血/再灌注损伤不同阶段表达的关键基因。
Neural Regen Res. 2016 Nov;11(11):1824-1829. doi: 10.4103/1673-5374.194754.
9
Electroacupuncture pretreatment attenuates spinal cord ischemia-reperfusion injury via inhibition of high-mobility group box 1 production in a LXA receptor-dependent manner.电针预处理通过以脂氧素A受体依赖性方式抑制高迁移率族蛋白B1的产生来减轻脊髓缺血再灌注损伤。
Brain Res. 2017 Mar 15;1659:113-120. doi: 10.1016/j.brainres.2017.01.008. Epub 2017 Jan 12.
10
The Pathway of Let-7a-1/2-3p and HMGB1 Mediated Dexmedetomidine Inhibiting Microglia Activation in Spinal Cord Ischemia-Reperfusion Injury Mice.Let-7a-1/2-3p 和高迁移率族蛋白 B1 介导的右美托咪定抑制脊髓缺血再灌注损伤小鼠小胶质细胞激活的途径。
J Mol Neurosci. 2019 Sep;69(1):106-114. doi: 10.1007/s12031-019-01338-4. Epub 2019 Jun 12.

引用本文的文献

1
Role of exercise on ncRNAs and exosomal ncRNAs in preventing neurodegenerative diseases: a narrative review.运动对非编码RNA和外泌体非编码RNA在预防神经退行性疾病中的作用：一项叙述性综述
Mol Med. 2025 Feb 7;31(1):51. doi: 10.1186/s10020-025-01091-y.
2
MiR129-5p-loaded exosomes suppress seizure-associated neurodegeneration in status epilepticus model mice by inhibiting HMGB1/TLR4-mediated neuroinflammation.载 miR129-5p 的外泌体通过抑制 HMGB1/TLR4 介导的神经炎症抑制癫痫持续状态模型小鼠的癫痫相关神经退行性变。
Mol Biol Rep. 2024 Feb 8;51(1):292. doi: 10.1007/s11033-024-09215-z.
3
The Role of Non-Coding RNAs in the Pathogenesis of Parkinson's Disease: Recent Advancement.

本文引用的文献

1
Elevated microRNA-129-5p level ameliorates neuroinflammation and blood-spinal cord barrier damage after ischemia-reperfusion by inhibiting HMGB1 and the TLR3-cytokine pathway.高表达 microRNA-129-5p 通过抑制 HMGB1 和 TLR3-细胞因子通路减轻缺血再灌注后神经炎症和血脊髓屏障损伤。
J Neuroinflammation. 2017 Oct 23;14(1):205. doi: 10.1186/s12974-017-0977-4.
非编码RNA在帕金森病发病机制中的作用：最新进展
Pharmaceuticals (Basel). 2022 Jun 30;15(7):811. doi: 10.3390/ph15070811.