Metabolically stressed adipocytes: mediators of cardioprotection via extracellular vesicle-mediated transport of oxidatively damaged mitochondria.
作者信息
Anthony Sarah R, Tranter Michael
机构信息
Division of Cardiovascular Health and Disease, Department of Internal Medicine, University of Cincinnati College of Medicine, Cincinnati, OH, USA.
出版信息
ExRNA. 2022 Jan;4. doi: 10.21037/exrna-21-31. Epub 2022 Jan 25.
Abstract
摘要
相似文献
1
Metabolically stressed adipocytes: mediators of cardioprotection via extracellular vesicle-mediated transport of oxidatively damaged mitochondria.代谢应激的脂肪细胞：通过细胞外囊泡介导的氧化损伤线粒体转运实现心脏保护的介质。
ExRNA. 2022 Jan;4. doi: 10.21037/exrna-21-31. Epub 2022 Jan 25.
2
Complex I within oxidatively stressed bovine heart mitochondria is glutathionylated on Cys-531 and Cys-704 of the 75-kDa subunit: potential role of CYS residues in decreasing oxidative damage.氧化应激状态下牛心脏线粒体中的复合物I在75 kDa亚基的半胱氨酸-531和半胱氨酸-704上发生谷胱甘肽化修饰：半胱氨酸残基在减少氧化损伤中的潜在作用
J Biol Chem. 2008 Sep 5;283(36):24801-15. doi: 10.1074/jbc.M803432200. Epub 2008 Jul 8.
3
Extracellular vesicle-mediated long-range communication in stressed retinal pigment epithelial cell monolayers.应激状态下人视网膜色素上皮细胞单层间细胞外囊泡介导的长距离通讯
Biochim Biophys Acta Mol Basis Dis. 2018 Aug;1864(8):2610-2622. doi: 10.1016/j.bbadis.2018.04.016. Epub 2018 Apr 21.
4
Y RNA fragment in extracellular vesicles confers cardioprotection via modulation of IL-10 expression and secretion.细胞外囊泡中的Y RNA片段通过调节白细胞介素-10的表达和分泌赋予心脏保护作用。
EMBO Mol Med. 2017 Mar;9(3):337-352. doi: 10.15252/emmm.201606924.
5
Stressed-out mitochondria get MAD.压力过大的线粒体变得疯狂。
Cell Metab. 2010 Dec 1;12(6):559-60. doi: 10.1016/j.cmet.2010.11.018.
6
Mechanisms of extracellular vesicle uptake in stressed retinal pigment epithelial cell monolayers.应激状态下人视网膜色素上皮细胞单层细胞中外泌体摄取的机制。
Biochim Biophys Acta Mol Basis Dis. 2020 Mar 1;1866(3):165608. doi: 10.1016/j.bbadis.2019.165608. Epub 2019 Nov 15.
7
Extracellular Cl-free-induced cardioprotection against hypoxia/reoxygenation is associated with attenuation of mitochondrial permeability transition pore.细胞外无 Cl-诱导的心肌缺氧/复氧保护作用与线粒体通透性转换孔的抑制有关。
Biomed Pharmacother. 2017 Feb;86:637-644. doi: 10.1016/j.biopha.2016.12.048. Epub 2016 Dec 26.
8
Proteolytic cleavage of cellubrevin and vesicle-associated membrane protein (VAMP) by tetanus toxin does not impair insulin-stimulated glucose transport or GLUT4 translocation in rat adipocytes.破伤风毒素对细胞ubrevin和囊泡相关膜蛋白（VAMP）的蛋白水解切割不会损害大鼠脂肪细胞中胰岛素刺激的葡萄糖转运或GLUT4易位。
Biochem J. 1997 Jan 1;321 ( Pt 1)(Pt 1):233-8. doi: 10.1042/bj3210233.
9
Modulation of Ca(2+)-dependent currents in metabolically stressed cultured sensory neurones by intracellular photorelease of ATP.通过细胞内光释放ATP对代谢应激培养的感觉神经元中钙依赖电流的调节
Br J Pharmacol. 1995 Jan;114(2):544-50. doi: 10.1111/j.1476-5381.1995.tb13261.x.
10
Helicobacter pylori infection is associated with oxidatively damaged DNA in human leukocytes and decreased level of urinary 8-oxo-7,8-dihydroguanine.幽门螺杆菌感染与人类白细胞中氧化损伤的DNA以及尿8-氧代-7,8-二氢鸟嘌呤水平降低有关。
Carcinogenesis. 2006 Mar;27(3):405-8. doi: 10.1093/carcin/bgi238. Epub 2005 Oct 11.
本文引用的文献
1
Exosomes from adipose-derived stem cells alleviate myocardial infarction via microRNA-31/FIH1/HIF-1α pathway.脂肪来源干细胞来源的外泌体通过 microRNA-31/FIH1/HIF-1α 通路减轻心肌梗死。
J Mol Cell Cardiol. 2022 Jan;162:10-19. doi: 10.1016/j.yjmcc.2021.08.010. Epub 2021 Aug 30.
2
Extracellular vesicle-based interorgan transport of mitochondria from energetically stressed adipocytes.基于细胞外囊泡的从能量应激脂肪细胞到其他器官的线粒体的转运。
Cell Metab. 2021 Sep 7;33(9):1853-1868.e11. doi: 10.1016/j.cmet.2021.08.002. Epub 2021 Aug 20.
3
Adipocyte-specific deletion of HuR induces spontaneous cardiac hypertrophy and fibrosis.脂肪细胞特异性敲除 HuR 诱导自发性心肌肥厚和纤维化。
Am J Physiol Heart Circ Physiol. 2021 Jul 1;321(1):H228-H241. doi: 10.1152/ajpheart.00957.2020. Epub 2021 May 21.
4
Adipose-Derived Mesenchymal Stem Cells-Derived Exosomes Carry MicroRNA-671 to Alleviate Myocardial Infarction Through Inactivating the TGFBR2/Smad2 Axis.脂肪间充质干细胞衍生的外泌体通过携带 microRNA-671 来抑制 TGFBR2/Smad2 轴缓解心肌梗死。
Inflammation. 2021 Oct;44(5):1815-1830. doi: 10.1007/s10753-021-01460-9. Epub 2021 Apr 21.
5
Molecular Mechanisms of Obesity-Linked Cardiac Dysfunction: An Up-Date on Current Knowledge.肥胖相关心脏功能障碍的分子机制：当前知识的更新。
Cells. 2021 Mar 12;10(3):629. doi: 10.3390/cells10030629.
6
Extracellular Vesicles From Epicardial Fat Facilitate Atrial Fibrillation.来自心外膜脂肪的细胞外囊泡促进心房颤动。
Circulation. 2021 Jun 22;143(25):2475-2493. doi: 10.1161/CIRCULATIONAHA.120.052009. Epub 2021 Apr 1.
7
MicroRNA-221/222 Mediates ADSC-Exosome-Induced Cardioprotection Against Ischemia/Reperfusion by Targeting PUMA and ETS-1.微小RNA-221/222通过靶向PUMA和ETS-1介导脂肪干细胞外泌体对缺血/再灌注损伤的心脏保护作用。
Front Cell Dev Biol. 2020 Dec 3;8:569150. doi: 10.3389/fcell.2020.569150. eCollection 2020.
8
Brown adipose tissue-derived exosomes mitigate the metabolic syndrome in high fat diet mice.棕色脂肪组织来源的外泌体可减轻高脂饮食小鼠的代谢综合征。
Theranostics. 2020 Jul 9;10(18):8197-8210. doi: 10.7150/thno.43968. eCollection 2020.
9
A Novel Model of Diabetic Complications: Adipocyte Mitochondrial Dysfunction Triggers Massive β-Cell Hyperplasia.一种新型糖尿病并发症模型：脂肪细胞线粒体功能障碍引发大量β细胞增生。
Diabetes. 2020 Mar;69(3):313-330. doi: 10.2337/db19-0327. Epub 2019 Dec 27.
10
Mechanisms linking adipose tissue inflammation to cardiac hypertrophy and fibrosis.脂肪组织炎症与心脏肥大和纤维化的关联机制。
Clin Sci (Lond). 2019 Nov 29;133(22):2329-2344. doi: 10.1042/CS20190578.
Zotero 插件