Becker C D, Scheidegger J, Marincek B
Gastrointest Radiol. 1986;11(4):305-11. doi: 10.1007/BF02035097.
Hepatic vein occlusion causes morphologic changes that can be demonstrated by computed tomography (CT) and ultrasound. In this study the imaging findings of acute, subacute, and chronic occlusion of the hepatic veins were analyzed retrospectively in 9 patients and correlated with the histopathologic changes. The CT findings were focal or scattered hypodense parenchymal lesions of the liver before and a patchy enhancement after intravenous bolus injection of contrast material. In none of the cases could the hepatic veins be identified. Hepatomegaly with relative enlargement of the caudate lobe was almost always observed. Ultrasonography demonstrated solid material within the major hepatic veins, intrahepatic venous collaterals, and focal parenchymal lesions, which varied with the stage of the disease: a hypoechogenic area was observed in acute hepatic vein thrombosis with subsequent hemorrhagic infarction; hyperechogenic lesions corresponded with fibrotic zones in chronic disease. Ascites was shown by both methods in all patients.
肝静脉闭塞会导致形态学改变，这些改变可通过计算机断层扫描（CT）和超声检查显示出来。在本研究中，对9例肝静脉急性、亚急性和慢性闭塞的影像学表现进行了回顾性分析，并与组织病理学改变进行了关联。CT表现为肝脏实质内局灶性或散在性低密度病变，静脉推注造影剂后呈斑片状强化。所有病例均无法识别肝静脉。几乎总是观察到肝脏肿大伴尾状叶相对增大。超声检查显示肝内主要静脉内有实性物质、肝内静脉侧支循环和局灶性实质病变，这些病变随疾病阶段而异：急性肝静脉血栓形成伴随后发性出血性梗死时可见低回声区；慢性疾病中的高回声病变与纤维化区域相对应。两种检查方法均显示所有患者有腹水。
