Spicer L J
Life Sci. 1986 Nov 10;39(19):1701-11. doi: 10.1016/0024-3205(86)90088-3.
A review of the rapidly accumulating data in the literature continues to support the notion that catecholamines regulate ovarian function, and extends the complexity of catecholaminergic effects on the ovary via interactions with pituitary and adrenal hormones. It is clear that catecholamines affect growth and differentiation of ovarian follicles, but their role in follicular rupture during ovulation and in corpus luteum function remains unclear. The effects of catecholamines (mediated by membrane receptors) on ovarian function probably should be considered paracrine but classic endocrine regulation of ovarian function cannot be ruled out. Myogenic tonus of ovarian vasculature appears to be regulated by catecholamines, and estrogens may enhance adrenergic receptors in ovarian smooth muscle cells.
对文献中迅速积累的数据进行的综述继续支持儿茶酚胺调节卵巢功能这一观点,并通过与垂体和肾上腺激素的相互作用扩展了儿茶酚胺能对卵巢影响的复杂性。很明显,儿茶酚胺会影响卵巢卵泡的生长和分化,但其在排卵期间卵泡破裂及黄体功能中的作用仍不清楚。儿茶酚胺(通过膜受体介导)对卵巢功能的影响可能应被视为旁分泌,但卵巢功能的经典内分泌调节也不能排除。卵巢血管的肌源性紧张似乎受儿茶酚胺调节,并且雌激素可能会增强卵巢平滑肌细胞中的肾上腺素能受体。