Retraction: Prenatal Cocaine Exposure Increases Synaptic Localization of a Neuronal RasGEF, GRASP-1 via Hyperphosphorylation of AMPAR Anchoring Protein, GRIP.
出版信息
PLoS One. 2022 Mar 30;17(3):e0266630. doi: 10.1371/journal.pone.0266630. eCollection 2022.
Abstract
摘要
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1
Retraction: Prenatal Cocaine Exposure Increases Synaptic Localization of a Neuronal RasGEF, GRASP-1 via Hyperphosphorylation of AMPAR Anchoring Protein, GRIP.撤稿声明：产前可卡因暴露通过使AMPA受体锚定蛋白GRIP过度磷酸化，增加神经元Ras鸟苷酸交换因子GRASP-1的突触定位。
PLoS One. 2022 Mar 30;17(3):e0266630. doi: 10.1371/journal.pone.0266630. eCollection 2022.
2
Prenatal cocaine exposure increases synaptic localization of a neuronal RasGEF, GRASP-1 via hyperphosphorylation of AMPAR anchoring protein, GRIP.产前可卡因暴露通过过度磷酸化 AMPAR 锚定蛋白 GRIP，增加神经元 RasGEF、GRASP-1 的突触定位。
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Prenatal cocaine reduces AMPA receptor synaptic expression through hyperphosphorylation of the synaptic anchoring protein GRIP.产前接触可卡因会通过突触锚定蛋白GRIP的过度磷酸化降低AMPA受体的突触表达。
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Physiological and pathological caspase cleavage of the neuronal RasGEF GRASP-1 as detected using a cleavage site-specific antibody.使用切割位点特异性抗体检测到的神经元Ras鸟苷酸交换因子GRASP-1的生理和病理半胱天冬酶切割。
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本文引用的文献
1
Prenatal Cocaine Exposure Upregulates BDNF-TrkB Signaling.产前可卡因暴露上调脑源性神经营养因子-酪氨酸激酶受体B（BDNF-TrkB）信号通路。
PLoS One. 2016 Aug 5;11(8):e0160585. doi: 10.1371/journal.pone.0160585. eCollection 2016.
2
Prenatal cocaine exposure uncouples mGluR1 from Homer1 and Gq Proteins.产前可卡因暴露使代谢型谷氨酸受体1（mGluR1）与亲代谢性谷氨酸受体结合蛋白1（Homer1）和Gq蛋白解偶联。
PLoS One. 2014 Mar 13;9(3):e91671. doi: 10.1371/journal.pone.0091671. eCollection 2014.
3
Prenatal cocaine exposure increases synaptic localization of a neuronal RasGEF, GRASP-1 via hyperphosphorylation of AMPAR anchoring protein, GRIP.产前可卡因暴露通过过度磷酸化 AMPAR 锚定蛋白 GRIP，增加神经元 RasGEF、GRASP-1 的突触定位。
PLoS One. 2011;6(9):e25019. doi: 10.1371/journal.pone.0025019. Epub 2011 Sep 27.
4
Naloxone's pentapeptide binding site on filamin A blocks Mu opioid receptor-Gs coupling and CREB activation of acute morphine.纳洛酮在细丝蛋白A上的五肽结合位点可阻断急性吗啡的μ阿片受体-Gs偶联及CREB激活。
PLoS One. 2009;4(1):e4282. doi: 10.1371/journal.pone.0004282. Epub 2009 Jan 27.
5
High-affinity naloxone binding to filamin a prevents mu opioid receptor-Gs coupling underlying opioid tolerance and dependence.高亲和力的纳洛酮与细丝蛋白A结合可防止μ阿片受体与Gs偶联，而这种偶联是阿片类药物耐受性和依赖性的基础。
PLoS One. 2008 Feb 6;3(2):e1554. doi: 10.1371/journal.pone.0001554.
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