• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

相似文献

1
Toll-Interacting Protein and Altered Lung Microbiota in Idiopathic Pulmonary Fibrosis.Toll相互作用蛋白与特发性肺纤维化中肺微生物群的改变
Am J Respir Crit Care Med. 2022 Jul 15;206(2):224-227. doi: 10.1164/rccm.202111-2590LE.
2
Lung Microbiota Contribute to Pulmonary Inflammation and Disease Progression in Pulmonary Fibrosis.肺部微生物组促进肺纤维化中的肺部炎症和疾病进展。
Am J Respir Crit Care Med. 2019 May 1;199(9):1127-1138. doi: 10.1164/rccm.201809-1650OC.
3
Dysregulated Lung Commensal Bacteria Drive Interleukin-17B Production to Promote Pulmonary Fibrosis through Their Outer Membrane Vesicles.失调的肺部共生菌通过其外膜囊泡产生白细胞介素-17B 以促进肺纤维化。
Immunity. 2019 Mar 19;50(3):692-706.e7. doi: 10.1016/j.immuni.2019.02.001. Epub 2019 Feb 26.
4
Potential targeted therapy based on deep insight into the relationship between the pulmonary microbiota and immune regulation in lung fibrosis.基于对肺纤维化中肺部微生物组与免疫调节关系的深入了解的潜在靶向治疗。
Front Immunol. 2023 Jan 24;14:1032355. doi: 10.3389/fimmu.2023.1032355. eCollection 2023.
5
Radiographic Honeycombing and Altered Lung Microbiota in Patients with Idiopathic Pulmonary Fibrosis.特发性肺纤维化患者的影像学蜂窝状改变与肺微生物群变化
Am J Respir Crit Care Med. 2019 Dec 15;200(12):1544-1547. doi: 10.1164/rccm.201903-0680LE.
6
Making the case for causality: what role do lung microbiota play in idiopathic pulmonary fibrosis?论证因果关系:肺部微生物群在特发性肺纤维化中起什么作用?
Eur Respir J. 2020 Apr 3;55(4). doi: 10.1183/13993003.00318-2020. Print 2020 Apr.
7
Inhibition of lung microbiota-derived proapoptotic peptides ameliorates acute exacerbation of pulmonary fibrosis.抑制肺微生物群衍生的促凋亡肽可改善肺纤维化的急性加重。
Nat Commun. 2022 Mar 23;13(1):1558. doi: 10.1038/s41467-022-29064-3.
8
Lung microbiome and disease progression in idiopathic pulmonary fibrosis: an analysis of the COMET study.肺微生物组与特发性肺纤维化疾病进展:COMET 研究分析。
Lancet Respir Med. 2014 Jul;2(7):548-56. doi: 10.1016/S2213-2600(14)70069-4. Epub 2014 Apr 21.
9
Measurement of hypoxia in the lung in idiopathic pulmonary fibrosis: a matter of control.特发性肺纤维化中肺部缺氧的测量:控制问题。
Eur Respir J. 2022 Mar 10;59(3). doi: 10.1183/13993003.02711-2021. Print 2022 Mar.
10
Reply: Measurement of hypoxia in the lung in idiopathic pulmonary fibrosis: a matter of control.回复:特发性肺纤维化中肺部缺氧的测量:控制问题。
Eur Respir J. 2022 Mar 10;59(3). doi: 10.1183/13993003.03124-2021. Print 2022 Mar.

引用本文的文献

1
Residual interstitial lung abnormalities post-COVID-19 infection appear independent of lung microbiota.新冠病毒感染后残留的间质性肺异常似乎与肺部微生物群无关。
ERJ Open Res. 2025 May 27;11(3). doi: 10.1183/23120541.01153-2024. eCollection 2025 May.
2
IL-10 sensing by lung interstitial macrophages prevents bacterial dysbiosis-driven pulmonary inflammation and maintains immune homeostasis.肺间质巨噬细胞对白细胞介素-10的感知可预防细菌失调驱动的肺部炎症并维持免疫稳态。
Immunity. 2025 May 13;58(5):1306-1326.e7. doi: 10.1016/j.immuni.2025.04.004. Epub 2025 Apr 29.
3
The lung microbiome in interstitial lung disease.间质性肺疾病中的肺部微生物群。
Breathe (Sheff). 2025 Apr 17;21(2):240167. doi: 10.1183/20734735.0167-2024. eCollection 2025 Apr.
4
Leveraging Microbiome Composition Variability for Precision Medicine in Idiopathic Pulmonary Fibrosis.利用微生物组组成变异性进行特发性肺纤维化的精准医学
Am J Respir Crit Care Med. 2024 Aug 15;210(4):527-528. doi: 10.1164/rccm.202310-1899LE.
5
Commensal Oral Microbiota, Disease Severity, and Mortality in Fibrotic Lung Disease.共生口腔微生物群、纤维化肺部疾病的疾病严重程度和死亡率。
Am J Respir Crit Care Med. 2024 May 1;209(9):1101-1110. doi: 10.1164/rccm.202308-1357OC.

本文引用的文献

1
Toll-like receptors, environmental caging, and lung dysbiosis. toll 样受体、环境笼养和肺部菌群失调。
Am J Physiol Lung Cell Mol Physiol. 2021 Aug 1;321(2):L404-L415. doi: 10.1152/ajplung.00002.2021. Epub 2021 Jun 23.
2
Functional lower airways genomic profiling of the microbiome to capture active microbial metabolism.对微生物组进行功能性下呼吸道基因组分析以捕获活跃的微生物代谢。
Eur Respir J. 2021 Jul 29;58(1). doi: 10.1183/13993003.03434-2020. Print 2021 Jul.
3
Toll interacting protein protects bronchial epithelial cells from bleomycin-induced apoptosis.Toll 相互作用蛋白可保护支气管上皮细胞免受博来霉素诱导的凋亡。
FASEB J. 2020 Aug;34(8):9884-9898. doi: 10.1096/fj.201902636RR. Epub 2020 Jun 28.
4
Toll-like Receptors and the Control of Immunity. toll 样受体与免疫的调控。
Cell. 2020 Mar 19;180(6):1044-1066. doi: 10.1016/j.cell.2020.02.041. Epub 2020 Mar 11.
5
Radiographic Honeycombing and Altered Lung Microbiota in Patients with Idiopathic Pulmonary Fibrosis.特发性肺纤维化患者的影像学蜂窝状改变与肺微生物群变化
Am J Respir Crit Care Med. 2019 Dec 15;200(12):1544-1547. doi: 10.1164/rccm.201903-0680LE.
6
Lung Microbiota Contribute to Pulmonary Inflammation and Disease Progression in Pulmonary Fibrosis.肺部微生物组促进肺纤维化中的肺部炎症和疾病进展。
Am J Respir Crit Care Med. 2019 May 1;199(9):1127-1138. doi: 10.1164/rccm.201809-1650OC.
7
Microbes Are Associated with Host Innate Immune Response in Idiopathic Pulmonary Fibrosis.微生物与特发性肺纤维化中的宿主固有免疫反应相关。
Am J Respir Crit Care Med. 2017 Jul 15;196(2):208-219. doi: 10.1164/rccm.201607-1525OC.
8
The role of bacteria in the pathogenesis and progression of idiopathic pulmonary fibrosis.细菌在特发性肺纤维化发病机制及进展中的作用。
Am J Respir Crit Care Med. 2014 Oct 15;190(8):906-13. doi: 10.1164/rccm.201403-0541OC.
9
Lung microbiome and disease progression in idiopathic pulmonary fibrosis: an analysis of the COMET study.肺微生物组与特发性肺纤维化疾病进展:COMET 研究分析。
Lancet Respir Med. 2014 Jul;2(7):548-56. doi: 10.1016/S2213-2600(14)70069-4. Epub 2014 Apr 21.
10
Genetic variants associated with idiopathic pulmonary fibrosis susceptibility and mortality: a genome-wide association study.与特发性肺纤维化易感性和死亡率相关的遗传变异:全基因组关联研究。
Lancet Respir Med. 2013 Jun;1(4):309-317. doi: 10.1016/S2213-2600(13)70045-6. Epub 2013 Apr 17.

Toll-Interacting Protein and Altered Lung Microbiota in Idiopathic Pulmonary Fibrosis.

作者信息

Lipinski Jay H, Erb-Downward John R, Huffnagle Gary B, Flaherty Kevin R, Martinez Fernando J, Moore Bethany B, Dickson Robert P, Noth Imre, O'Dwyer David N

机构信息

University of Michigan Ann Arbor, Michigan.

Cornell University New York, New York.

出版信息

Am J Respir Crit Care Med. 2022 Jul 15;206(2):224-227. doi: 10.1164/rccm.202111-2590LE.

DOI:10.1164/rccm.202111-2590LE
PMID:35446241
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9887421/
Abstract
摘要