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The KN-93 Molecule Inhibits Calcium/Calmodulin-Dependent Protein Kinase II (CaMKII) Activity by Binding to Ca/CaM.
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Physical interactions driving the activation/inhibition of calcium/calmodulin dependent protein kinase II.
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Further Evidence of the Melatonin Calmodulin Interaction: Effect on CaMKII Activity.
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Phosphorylation status of the NR2B subunit of NMDA receptor regulates its interaction with calcium/calmodulin-dependent protein kinase II.
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Distinct roles of calmodulin and Ca/calmodulin-dependent protein kinase II in isopreterenol-induced cardiac hypertrophy.
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Cardioprotective effects of naringin in a type 2 diabetes rodent model by reducing calcium overload and oxidative stress.
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The role of stem cell-derived exosomes in regulating pyroptosis for disease therapy.
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Ca/calmodulin-dependent protein kinase II regulates the inflammatory hDPSCs dentino-differentiation via BDNF/TrkB receptor signaling.
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The Effect of Selected Nitric Oxide Synthase Polymorphisms on the Risk of Developing Diabetic Nephropathy.
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Glucose fluctuations aggravate myocardial fibrosis via activating the CaMKII/Stat3 signaling in type 2 diabtetes.
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The Effect of Omega-3 Fatty Acids on Insulin Resistance.
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Adipocyte CAMK2 deficiency improves obesity-associated glucose intolerance.
Mol Metab. 2021 Nov;53:101300. doi: 10.1016/j.molmet.2021.101300. Epub 2021 Jul 22.
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Multiple Roles of 25-Hydroxycholesterol in Lipid Metabolism, Antivirus Process, Inflammatory Response, and Cell Survival.
Oxid Med Cell Longev. 2020 Nov 19;2020:8893305. doi: 10.1155/2020/8893305. eCollection 2020.
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CaM Kinase II-δ Is Required for Diabetic Hyperglycemia and Retinopathy but Not Nephropathy.
Diabetes. 2021 Feb;70(2):616-626. doi: 10.2337/db19-0659. Epub 2020 Nov 25.
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Glucagon stimulates gluconeogenesis by INSP3R1-mediated hepatic lipolysis.
Nature. 2020 Mar;579(7798):279-283. doi: 10.1038/s41586-020-2074-6. Epub 2020 Mar 4.
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Distinct Types of Cell Death and the Implication in Diabetic Cardiomyopathy.
Front Pharmacol. 2020 Feb 7;11:42. doi: 10.3389/fphar.2020.00042. eCollection 2020.
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