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本文引用的文献
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Platelet-monocyte interaction amplifies thromboinflammation through tissue factor signaling in COVID-19.
Blood Adv. 2022 Sep 13;6(17):5085-5099. doi: 10.1182/bloodadvances.2021006680.
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Effect of P2Y12 Inhibitors on Survival Free of Organ Support Among Non-Critically Ill Hospitalized Patients With COVID-19: A Randomized Clinical Trial.
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COVID-19 and antiphospholipid antibodies: A position statement and management guidance from AntiPhospholipid Syndrome Alliance for Clinical Trials and InternatiOnal Networking (APS ACTION).
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Severity of SARS-CoV-2 infection and albumin levels recorded at the first emergency department evaluation: a multicentre retrospective observational study.
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Platelets amplify endotheliopathy in COVID-19.
Sci Adv. 2021 Sep 10;7(37):eabh2434. doi: 10.1126/sciadv.abh2434. Epub 2021 Sep 8.
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Thromboembolic Complications in Covid-19: From Clinical Scenario to Laboratory Evidence.
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Pathophysiologic mechanisms of cerebral endotheliopathy and stroke due to Sars-CoV-2.
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The interplay between neutrophils, complement, and microthrombi in COVID-19.
Best Pract Res Clin Rheumatol. 2021 Mar;35(1):101661. doi: 10.1016/j.berh.2021.101661. Epub 2021 Jan 13.
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Flow-Mediated Susceptibility and Molecular Response of Cerebral Endothelia to SARS-CoV-2 Infection.
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Is there a role for the ACE2 receptor in SARS-CoV-2 interactions with platelets?
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