血管活性肠肽和卡巴胆碱协同作用，诱导大鼠颈上神经节中含肌醇磷脂的水解。
Vasoactive intestinal polypeptide and carbachol act synergistically to induce the hydrolysis of inositol containing phospholipids in the rat superior cervical ganglion.
作者信息
Durroux T, Barberis C, Jard S
出版信息
Neurosci Lett. 1987 Mar 31;75(2):211-5. doi: 10.1016/0304-3940(87)90299-0.
PMID:3574774
Abstract
The effects of vasoactive intestinal polypeptide (VIP) and carbachol on inositol lipid breakdown were assayed in isolated rat superior cervical ganglia. We report here that VIP and carbachol act synergistically to stimulate the formation of inositol phosphates. This synergistic interaction may explain the modulatory effect of VIP on the muscarinic transmission in the sympathetic ganglia.
摘要
在离体大鼠颈上神经节中检测了血管活性肠肽（VIP）和卡巴胆碱对肌醇脂质分解的影响。我们在此报告，VIP和卡巴胆碱协同作用以刺激肌醇磷酸的形成。这种协同相互作用可能解释了VIP对交感神经节中毒蕈碱传递的调节作用。
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