Fukamachi A, Horikoshi T, Nagaseki Y, Sasaki H, Nukui H
Acta Neurochir (Wien). 1987;84(3-4):89-92. doi: 10.1007/BF01418830.
Three patients with symmetrical bilateral low-density areas which were the perfusion territories of Heubner's arteries are reported. The infarctions were demonstrated on computed tomography after aneurysm surgery. Two of them had an anterior communicating artery aneurysm and the other one had multiple aneurysms of both sides. They were operated upon in the acute stage after subarachnoid haemorrhage. As common factors, a thick and diffuse subarachnoid haemorrhage, an acute-stage operation, temporary clips to bilateral A1 segments or the internal carotid artery and a relatively low systemic blood pressure during operation were found. From these findings we conclude that the pathogenesis of the infarctions was due to occlusive changes affecting Heubner's arteries bilaterally.
本文报告了3例双侧对称性低密度区患者，这些区域是Heubner动脉的灌注区。动脉瘤手术后的计算机断层扫描显示了梗死灶。其中2例患有前交通动脉瘤，另1例双侧有多个动脉瘤。他们在蛛网膜下腔出血后的急性期接受了手术。发现共同的因素有：浓厚且弥漫性的蛛网膜下腔出血、急性期手术、双侧A1段或颈内动脉的临时夹闭以及手术期间相对较低的全身血压。从这些发现中我们得出结论，梗死的发病机制是由于双侧Heubner动脉发生了闭塞性改变。
