Tomura Nobunari, Fujino Masashi, Kataoka Yu, Yoneda Shuichi, Sasaki Hiroaki, Noguchi Teruo
Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, 6-1 Kishibe-Shimmachi, Suita, Osaka, Japan.
Department of Cardiovascular Surgery, National Cerebral and Cardiovascular Center, 6-1 Kishibe-Shimmachi, Suita, Osaka, Japan.
Case Rep Cardiol. 2022 Jun 22;2022:7712888. doi: 10.1155/2022/7712888. eCollection 2022.
It is sometimes difficult to identify the culprit lesion and treatment strategy in patients with acute coronary syndrome who have complex coronary lesions and jeopardized left internal mammary artery graft. This report describes a heart team approach for a non-ST-segment elevation myocardial infarction case with complex coronary vasculature. A 73-year-old man presented to the emergency department with crescendo angina. He had a history of total aortic arch replacement with concomitant coronary artery bypass graft using left internal mammary artery. Emergent coronary angiography demonstrated severe stenosis at left main trunk bifurcation caused by calcified nodule. While the bypass graft to left anterior descending coronary artery was patent, the proximal segment of left subclavian artery was occluded. Following the prompt discussion with our heart team, we performed percutaneous coronary intervention in the first step for treating the left main stenosis using rotational atherectomy into the unprotected left circumflex artery. After clinical recovery, stress myocardial scintigraphy identified the presence of anteroseptal ischemia, which indicated coronary subclavian steal syndrome due to left subclavian artery occlusion. Contrast-enhanced CT visualized that the occlusion originated from the anastomosis, suggesting the potential procedural risk of endovascular treatment by dilatation. Our heart team discussed again and decided to undergo axillo-axillary artery bypass surgery. He was discharged 8 days after the surgery without any sequelae. This is the rare case report of non-ST-segment elevation myocardial infarction who had similar condition to coronary subclavian steal syndrome after total aortic arch replacement. This case highlights the importance of a collaborative approach of the heart team to identify the best therapeutic strategy in a patient with complex coronary vasculature.
对于患有复杂冠状动脉病变且左乳内动脉移植血管受到威胁的急性冠状动脉综合征患者,有时很难确定罪魁祸首病变和治疗策略。本报告描述了一种针对具有复杂冠状动脉血管系统的非ST段抬高型心肌梗死病例的心脏团队治疗方法。一名73岁男性因进行性加重的心绞痛就诊于急诊科。他有全主动脉弓置换术史,并同时使用左乳内动脉进行冠状动脉旁路移植术。急诊冠状动脉造影显示左主干分叉处因钙化结节导致严重狭窄。虽然左前降支冠状动脉的旁路移植血管通畅,但左锁骨下动脉近端段闭塞。在与我们的心脏团队进行迅速讨论后,我们首先进行了经皮冠状动脉介入治疗,使用旋磨术治疗未受保护的左旋支动脉的左主干狭窄。临床恢复后,负荷心肌闪烁显像显示前间隔缺血,提示由于左锁骨下动脉闭塞导致冠状动脉锁骨下窃血综合征。增强CT显示闭塞起源于吻合口,提示扩张性血管内治疗存在潜在的手术风险。我们的心脏团队再次讨论后决定进行腋-腋动脉搭桥手术。术后8天他出院,无任何后遗症。这是一例全主动脉弓置换术后出现与冠状动脉锁骨下窃血综合征类似情况的非ST段抬高型心肌梗死的罕见病例报告。该病例突出了心脏团队协作方法在确定具有复杂冠状动脉血管系统患者最佳治疗策略方面的重要性。