Souty-Grosset C, Juchault P
Gen Comp Endocrinol. 1987 May;66(2):163-70. doi: 10.1016/0016-6480(87)90264-4.
In some natural populations of Armadilidium vulgare, intersex animals are genetic males which are feminized by maternally transmitted symbiotic bacteria. In these intersex males (iM) the fat body synthesizes vitellogenin, although their gonads are testes with hypertrophied--but nonfunctional--androgenic glands. Vitellogenin is present in the hemolymph of males changed experimentally into iM 90 days after inoculation of the feminizing bacteria. During the molting cycle, vitellogenin synthesis in iM varies as in ovariectomized females or in vitellogenic females, with a peak at the stage D1." In A. vulgare, vitellogenin synthesis is a neutral character since it can be observed in a genetic male or in an ovariectomized female; however, it is inhibited by the androgenic hormone. In intersex males, vitellogenin synthesis is the result of their refractoriness to androgenic hormone.
在普通鼠妇的一些自然种群中，雌雄间体动物是遗传上的雄性，它们被母系传播的共生细菌雌性化。在这些雌雄间体雄性（iM）中，脂肪体合成卵黄蛋白原，尽管它们的性腺是睾丸，且具有肥大但无功能的雄激素腺。在接种雌性化细菌90天后通过实验转变为iM的雄性血淋巴中存在卵黄蛋白原。在蜕皮周期中，iM中的卵黄蛋白原合成与去卵巢雌性或卵黄生成期雌性一样变化，在D1阶段达到峰值。在普通鼠妇中，卵黄蛋白原合成是一个中性特征，因为它可以在遗传雄性或去卵巢雌性中观察到；然而，它会被雄激素抑制。在雌雄间体雄性中，卵黄蛋白原合成是它们对雄激素不敏感的结果。
