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视神经损伤伴长期失明及随后的视觉诱发电位

Optic nerve trauma with prolonged blindness followed by visual-evoked potential.

作者信息

Abraham F A, Spierer A, Blumenthal M

出版信息

Ophthalmologica. 1987;194(1):40-3. doi: 10.1159/000309732.

Abstract

An accidental deep intraorbital penetration of a radio antenna tip damaged the optic nerve and caused immediate amaurosis in a 19-year-old female with normal funduscopic, electroretinographic and computerized tomographic orbital findings. A small visual-evoked potential (VEP) recorded within 2 h of injury and in the following days revealed persistent electrical nerve conduction despite loss of vision and afferent pupillary reaction. The VEP constituted the only anticipating sign for the visual recovery which occurred after 3 days. Further increase of the VEP amplitudes preceded the visual improvement, resulting, finally, in 6/24 visual acuity with only lower nasal quadrant scotoma and slight optic disk pallor.

摘要

一名19岁女性,眼底、视网膜电图及眼眶计算机断层扫描结果均正常,因无线电天线尖端意外深部眶内刺入损伤视神经,导致立即失明。受伤后2小时内及随后几天记录的小视觉诱发电位(VEP)显示,尽管视力丧失和传入性瞳孔反应消失,但神经电传导持续存在。VEP是3天后出现视力恢复的唯一预示迹象。VEP振幅的进一步增加先于视力改善,最终视力达到6/24,仅存在鼻下象限小暗点和轻度视盘苍白。

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