[急性一氧化碳中毒后迟发性脑病发病机制的研究进展]
[Research progress on the mechanisms of delayed encephalopathy in acute carbon monoxide poisoning].
作者信息
Li C, Liang M L, Zhang X G
机构信息
Department of Poisoning and Occupational Diseases, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan 250021, China.
Department of Intensive Care Unit, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan 250021, China.
出版信息
Zhonghua Lao Dong Wei Sheng Zhi Ye Bing Za Zhi. 2022 Jul 20;40(7):543-546. doi: 10.3760/cma.j.cn121094-20210929-00480.
In our country, there are a large number of carbon monoxide poisoning patients every winter. 10% to 30% of patients with acute carbon monoxide poisoning develop acute carbon monoxide poisoning delayed encephalopathy after a "false recovery period" of about 2 to 60 days. The morbidity and mortality rates of the disease are extremely high, but there is still no effective treatment for this condition. The pathogenesis of the disease is complex, and there is no clear conclusion yet. After consulting a large number of recent relevant literatures, this article reviews the main research results of the pathogenesis of the disease so far, with an aim to facilitate its early clinical diagnosis and correct treatment.
在我国，每年冬季都有大量一氧化碳中毒患者。10%至30%的急性一氧化碳中毒患者在经过约2至60天的“假愈期”后会发生急性一氧化碳中毒迟发性脑病。该病的发病率和死亡率极高，但目前仍无有效的治疗方法。其发病机制复杂，尚无明确结论。本文查阅了大量近期相关文献，对目前该病发病机制的主要研究成果进行综述，旨在促进其早期临床诊断及正确治疗。
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