CD39/CD73失调与腺苷代谢促成了蕈样肉芽肿综合征患者的T细胞免疫抑制。 - Suppr | 超能文献

相似文献

1

CD39/CD73 dysregulation and adenosine metabolism contribute to T-cell immunosuppression in patients with Sézary syndrome.CD39/CD73失调与腺苷代谢促成了蕈样肉芽肿综合征患者的T细胞免疫抑制。

Blood. 2023 Jan 5;141(1):111-116. doi: 10.1182/blood.2022017259.

2

Synergy between the ectoenzymes CD39 and CD73 contributes to adenosinergic immunosuppression in human malignant gliomas.细胞外酶 CD39 和 CD73 的协同作用导致人恶性脑胶质瘤中的腺苷能免疫抑制。

Neuro Oncol. 2013 Sep;15(9):1160-72. doi: 10.1093/neuonc/not067. Epub 2013 Jun 4.

3

CD73 and CD39 ectonucleotidases in T cell differentiation: Beyond immunosuppression.T细胞分化中的CD73和CD39外核苷酸酶：超越免疫抑制

FEBS Lett. 2015 Nov 14;589(22):3454-60. doi: 10.1016/j.febslet.2015.07.027. Epub 2015 Jul 29.

4

CD39-CD73-adenosine effects in Sézary syndrome.CD39-CD73-腺苷在蕈样肉芽肿中的作用

Blood. 2023 Jan 5;141(1):9-10. doi: 10.1182/blood.2022018185.

5

Adenosine and the adenosine A2A receptor agonist, CGS21680, upregulate CD39 and CD73 expression through E2F-1 and CREB in regulatory T cells isolated from septic mice.腺苷及腺苷A2A受体激动剂CGS21680通过E2F-1和CREB上调从脓毒症小鼠分离出的调节性T细胞中CD39和CD73的表达。

Int J Mol Med. 2016 Sep;38(3):969-75. doi: 10.3892/ijmm.2016.2679. Epub 2016 Jul 14.

6

Involvement of the CD39/CD73/adenosine pathway in T-cell proliferation and NK cell-mediated antibody-dependent cell cytotoxicity in Sézary syndrome.CD39/CD73/腺苷通路在蕈样肉芽肿综合征中T细胞增殖及自然杀伤细胞介导的抗体依赖性细胞毒性中的作用

Blood. 2022 Apr 28;139(17):2712-2716. doi: 10.1182/blood.2021014782.

7

The role of the CD39-CD73-adenosine pathway in liver disease.CD39-CD73-腺苷通路在肝脏疾病中的作用。

J Cell Physiol. 2021 Feb;236(2):851-862. doi: 10.1002/jcp.29932. Epub 2020 Jul 10.

8

Conversion of ATP to adenosine by CD39 and CD73 in multiple myeloma can be successfully targeted together with adenosine receptor A2A blockade.在多发性骨髓瘤中，通过 CD39 和 CD73 将 ATP 转化为腺苷，可以与腺苷受体 A2A 阻断一起成功靶向。

J Immunother Cancer. 2020 May;8(1). doi: 10.1136/jitc-2020-000610.

9

Resident cardiac immune cells and expression of the ectonucleotidase enzymes CD39 and CD73 after ischemic injury.缺血损伤后心肌固有免疫细胞和胞外核苷酸酶 CD39 和 CD73 的表达。

PLoS One. 2012;7(4):e34730. doi: 10.1371/journal.pone.0034730. Epub 2012 Apr 13.

10

Ectonucleotidase CD73 and CD39 expression in non-small cell lung cancer relates to hypoxia and immunosuppressive pathways.非小细胞肺癌中核苷酸酶 CD73 和 CD39 的表达与缺氧和免疫抑制途径有关。

Life Sci. 2020 Oct 15;259:118389. doi: 10.1016/j.lfs.2020.118389. Epub 2020 Sep 6.

引用本文的文献

1

The CD39/CD73/Adenosine and NAD/CD38/CD203a/CD73 Axis in Cutaneous T-Cell Lymphomas.皮肤T细胞淋巴瘤中的CD39/CD73/腺苷与NAD/CD38/CD203a/CD73轴

Cells. 2025 Feb 19;14(4):309. doi: 10.3390/cells14040309.

2

Decoding NAD+ Metabolism in COVID-19: Implications for Immune Modulation and Therapy.解析新冠病毒感染中的NAD+代谢：对免疫调节和治疗的意义

Vaccines (Basel). 2024 Dec 24;13(1):1. doi: 10.3390/vaccines13010001.

3

Metabolomics analyses reveal the crucial role of ERK in regulating metabolic pathways associated with the proliferation of human cutaneous T-cell lymphoma cells treated with Glabridin.