Kuhlbusch R, Erckenbrecht J F
Leber Magen Darm. 1987 Jul;17(3):143-9.
Recto-anal continence results from a complex interplay of motor, sensory and anatomical continence mechanisms. Accordingly, fecal incontinence may be due to motor defects, sensory defects, neurological defects, or defects of the reservoir function of the ano-rectum. In a study in 16 patients with fecal incontinence and 16 continent controls the question was examined, to which extent disturbances of the motor function of the internal and external anal sphincter contribute to the pathogenesis of fecal incontinence. Basal and squeeze anal sphincter pressures were not different in incontinent and continent patients. However, 8/16 incontinent patients showed no or only an insufficient increase of external anal sphincter function while squeezing. It is concluded that disturbances of the motor function of the anal sphincters contribute to fecal incontinence in only about one half of the patients. In addition, the pressure increase of the sphincters during squeezing is of more importance for recto-anal continence than the absolute height of the muscular force of the anal sphincters.
直肠肛门节制功能源于运动、感觉和解剖学节制机制的复杂相互作用。因此，大便失禁可能是由于运动缺陷、感觉缺陷、神经缺陷或肛门直肠储器功能缺陷所致。在一项针对16例大便失禁患者和16例对照者的研究中，探讨了肛门内、外括约肌运动功能障碍在大便失禁发病机制中的作用程度。大便失禁患者和对照者的基础肛门括约肌压力和收缩时的肛门括约肌压力并无差异。然而，16例大便失禁患者中有8例在收缩时肛门外括约肌功能无增加或仅增加不足。得出的结论是，肛门括约肌运动功能障碍仅在约一半的患者中导致大便失禁。此外，收缩时括约肌的压力增加对直肠肛门节制功能的重要性超过肛门括约肌肌肉力量的绝对大小。
