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1
Interactions between uv radiation of different energies in the inactivation of bacteria.不同能量的紫外线辐射在细菌灭活过程中的相互作用。
J Bacteriol. 1978 Oct;136(1):437-40. doi: 10.1128/jb.136.1.437-440.1978.
2
Near-ultraviolet radiation blocks SOS responses to DNA damage in Escherichia coli.近紫外辐射可阻断大肠杆菌中对DNA损伤的SOS反应。
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Repair of near-ultraviolet (365 nm)-induced strand breaks in Escherichia coli DNA. The role of the polA and recA gene products.大肠杆菌DNA中近紫外线（365纳米）诱导的链断裂的修复。polA和recA基因产物的作用。
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Quantitative evidence for enzymatically-indeced DNA double-strand breaks as lethal lesions in UV irradiated pol+ and polA1 strains of E. coli K-12.紫外线照射的大肠杆菌K-12的pol+和polA1菌株中，酶促诱导的DNA双链断裂作为致死性损伤的定量证据。
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Calibration of optimal use parameters for an ultraviolet light-emitting diode in eliminating bacterial contamination on needleless connectors.用于消除无针连接器上细菌污染的紫外线发光二极管最佳使用参数的校准
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Influence of low-intensity ultraviolet radiation on extrusion of furanocoumarins to the leaf surface.低强度紫外线辐射对香豆素向叶片表面挤出的影响。
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Inactivation of bacterial pathogens following exposure to light from a 405-nanometer light-emitting diode array.暴露于405纳米发光二极管阵列发出的光后细菌病原体的失活
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4
Potential repair of Escherichia coli DNA following exposure to UV radiation from both medium- and low-pressure UV sources used in drinking water treatment.在饮用水处理中使用的中压和低压紫外线源照射后，大肠杆菌DNA的潜在修复。
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Lethal and mutational effects of solar and UV radiation on Staphylococcus aureus.太阳辐射和紫外线辐射对金黄色葡萄球菌的致死及诱变作用。
Arch Microbiol. 1992;157(3):242-8. doi: 10.1007/BF00245157.

本文引用的文献

1
DNA repair and genetic recombination: studies on mutants of Escherichia coli defective in these processes.DNA修复与基因重组：对在这些过程中存在缺陷的大肠杆菌突变体的研究。
Radiat Res. 1966:Suppl 6:156+.
2
Thymine-dimer and action-spectrum evidence for indirect photoreactivation in Escherichia coli.大肠杆菌中间接光复活作用的胸腺嘧啶二聚体及作用光谱证据。
Photochem Photobiol. 1969 Dec;10(6):383-95. doi: 10.1111/j.1751-1097.1969.tb05703.x.
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Additive erythemogenic effects of middle-(280-320 nm) and long-(320-400 nm) wave ultraviolet light.
J Invest Dermatol. 1974 Sep;63(3):273-8. doi: 10.1111/1523-1747.ep12680141.
4
Induction of single-strand breaks (alkali-labile bonds) in bacterial and phage DNA by near UV (365 nm) radiation.近紫外光（365纳米）辐射对细菌和噬菌体DNA中单链断裂（碱不稳定键）的诱导作用。
Photochem Photobiol. 1974 Nov;20(5):395-8. doi: 10.1111/j.1751-1097.1974.tb06593.x.
5
Reduced dimer excision in bacteria following near ultraviolet (365 nm) radiation.
Mutat Res. 1973 Sep;19(3):361-4. doi: 10.1016/0027-5107(73)90238-8.
6
Induction of pyrimidine dimers in bacterial DNA by 365 nm radiation.365纳米辐射诱导细菌DNA中的嘧啶二聚体。
Photochem Photobiol. 1973 Jan;17(1):69-73. doi: 10.1111/j.1751-1097.1973.tb06334.x.
7
Photoprotection from killing in ultraviolet-sensitive Escherichia coli K-12 mutants: involvement of excision-resynthesis repair.
Photochem Photobiol. 1972 Sep;16(3):197-202. doi: 10.1111/j.1751-1097.1972.tb06291.x.
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Evidence against DNA as the target for 334 nm-induced growth delay in Escherichia coli.
Photochem Photobiol. 1975 Apr;21(4):227-33. doi: 10.1111/j.1751-1097.1975.tb06661.x.
9
Synergistic lethal action of ultraviolet violet radiations and mild heat in Escherichia coli.
Photochem Photobiol. 1976 Oct;24(4):345-51. doi: 10.1111/j.1751-1097.1976.tb06835.x.
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Action spectra for lethality in recombination-less strains of Salmonella typhimurium and Escherichia coli.
Photochem Photobiol. 1976 Oct;24(4):337-43. doi: 10.1111/j.1751-1097.1976.tb06834.x.

不同能量的紫外线辐射在细菌灭活过程中的相互作用。

Interactions between uv radiation of different energies in the inactivation of bacteria.

作者信息

Tyrrell R M, Peak M J

出版信息

J Bacteriol. 1978 Oct;136(1):437-40. doi: 10.1128/jb.136.1.437-440.1978.

DOI:10.1128/jb.136.1.437-440.1978
PMID:361702
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC218678/
Abstract

A strong lethal interaction was observed between various monochromatic wavelengths (254, 334, 365, and 405 nm) in the repair-proficient E. coli K-12 strain AB 1157, except in the case of preexposure to 405-nm radiation which resulted in a protection against the inactivation resulting from subsequent exposure to 365-or 254-nm radiations. The results may be tentatively explained by assuming two classes of DNA lesions and two classes of damage to repair (reversible and inrreversible) whose proportions vary according to wavelength.

摘要

在修复能力正常的大肠杆菌K-12菌株AB 1157中，观察到各种单色波长（254、334、365和405纳米）之间存在强烈的致死相互作用，但预先暴露于405纳米辐射的情况除外，这种预先暴露可使细胞免受随后暴露于365纳米或254纳米辐射所导致的失活。这些结果可以通过假设两类DNA损伤和两类修复损伤（可逆和不可逆）来初步解释，其比例根据波长而变化。