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Initiation of the intrinsic coagulation and fibrinolytic pathways of man: the role of surfaces, hageman factor, prekallikrein, high molecular weight kininogen, and factor XI.

作者信息

Kaplan A P

出版信息

Prog Hemost Thromb. 1978;4:127-75.

PMID:362477
Abstract
摘要

相似文献

1
Initiation of the intrinsic coagulation and fibrinolytic pathways of man: the role of surfaces, hageman factor, prekallikrein, high molecular weight kininogen, and factor XI.人类内源性凝血和纤维蛋白溶解途径的启动:表面、哈格曼因子、前激肽释放酶、高分子量激肽原和因子XI的作用。
Prog Hemost Thromb. 1978;4:127-75.
2
[Initiation mechanism of blood coagulation (author's transl)].血液凝固的起始机制(作者译)
Tanpakushitsu Kakusan Koso. 1980 Feb;25(2):95-111.
3
Activation and function of human Hageman factor. The role of high molecular weight kininogen and prekallikrein.人凝血因子Ⅻ的激活与功能。高分子量激肽原和前激肽释放酶的作用。
J Clin Invest. 1977 Jul;60(1):18-31. doi: 10.1172/JCI108754.
4
Role of the contact system in fibrinolysis.接触系统在纤维蛋白溶解中的作用。
Semin Thromb Hemost. 1987 Jan;13(1):50-68. doi: 10.1055/s-2007-1003475.
5
Biochemical and functional properties of factor XI and prekallikrein.因子XI和前激肽释放酶的生化及功能特性
Semin Thromb Hemost. 1987 Jan;13(1):25-35. doi: 10.1055/s-2007-1003473.
6
Interaction between factor XII (Hageman factor), high molecular weight kininogen and prekallikrein.凝血因子 XII(哈格曼因子)、高分子量激肽原和前激肽释放酶之间的相互作用。
Thromb Res. 1976 Nov;9(5):423-33. doi: 10.1016/0049-3848(76)90198-5.
7
Contact factors in health and disease.健康与疾病中的接触因子。
Semin Thromb Hemost. 1987 Jan;13(1):36-49. doi: 10.1055/s-2007-1003474.
8
The "contact system" in health and disease.健康与疾病中的“接触系统”
Adv Intern Med. 1980;25:217-38.
9
Role of high-molecular-weight kininogen in surface-binding and activation of coagulation Factor XI and prekallikrein.高分子量激肽原在凝血因子XI和前激肽释放酶的表面结合及激活中的作用。
Proc Natl Acad Sci U S A. 1977 Oct;74(10):4636-40. doi: 10.1073/pnas.74.10.4636.
10
Prekallikrein activation by Hageman factor: potentiation by proteins in the absence of high molecular weight kininogen.哈格曼因子对前激肽释放酶的激活:在缺乏高分子量激肽原的情况下蛋白质的增强作用。
Trans Assoc Am Physicians. 1979;92:208-17.

引用本文的文献

1
Moderation of prekallkrein-factor XII interactions in surface activation of coagulation by protein-adsorption competition.通过蛋白质吸附竞争对凝血表面激活中前激肽释放酶 - 因子XII相互作用的调节。
Biomaterials. 2009 Oct;30(28):4915-20. doi: 10.1016/j.biomaterials.2009.05.076. Epub 2009 Jun 23.
2
Contributions of contact activation pathways of coagulation factor XII in plasma.血浆中凝血因子XII接触激活途径的作用。
J Biomed Mater Res A. 2009 Jul;90(1):27-34. doi: 10.1002/jbm.a.32076.
3
Clot formation by group A streptococci.A组链球菌导致的血栓形成。
Infect Immun. 1998 May;66(5):2362-4. doi: 10.1128/IAI.66.5.2362-2364.1998.
4
Endotoxin-induced pulmonary dysfunction is prevented by C1-esterase inhibitor.C1酯酶抑制剂可预防内毒素诱导的肺功能障碍。
J Clin Invest. 1993 Jun;91(6):2754-60. doi: 10.1172/JCI116516.
5
Activation of the classical pathway of complement by Hageman factor fragment.凝血因子片段激活补体经典途径。
J Exp Med. 1981 Mar 1;153(3):665-76. doi: 10.1084/jem.153.3.665.
6
Purified human plasma kallikrein aggregates human blood neutrophils.纯化的人血浆激肽释放酶可使人类血液中的中性粒细胞聚集。
J Clin Invest. 1982 May;69(5):1199-202. doi: 10.1172/jci110557.
7
The activation of the alternative pathway C3 convertase by human plasma kallikrein.人血浆激肽释放酶对替代途径C3转化酶的激活作用。
Immunology. 1982 Mar;45(3):587-95.
8
Physiologic manifestations of human anaphylaxis.人类过敏反应的生理表现。
J Clin Invest. 1980 Nov;66(5):1072-80. doi: 10.1172/JCI109936.
9
Binding of factors IX and IXa to cultured vascular endothelial cells.凝血因子IX和IXa与培养的血管内皮细胞的结合。
Proc Natl Acad Sci U S A. 1983 Jul;80(13):4119-23. doi: 10.1073/pnas.80.13.4119.
10
Cleavage of human high molecular weight kininogen markedly enhances its coagulant activity. Evidence that this molecule exists as a procofactor.人高分子量激肽原的裂解显著增强其凝血活性。该分子作为前辅因子存在的证据。
J Clin Invest. 1984 Apr;73(4):954-62. doi: 10.1172/JCI111319.