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“死亡驱力”的科学再思考:并非一种驱力,而是一组创伤诱发的自我成瘾性疾病。

"Death drive" scientifically reconsidered: Not a drive but a collection of trauma-induced auto-addictive diseases.

作者信息

Kirsch Michael, Dimitrijevic Aleksandar, Buchholz Michael B

机构信息

Institute of Physiological Chemistry, University Hospital Essen, Essen, Germany.

International Psychoanalytic University, Berlin, Germany.

出版信息

Front Psychol. 2022 Sep 28;13:941328. doi: 10.3389/fpsyg.2022.941328. eCollection 2022.

Abstract

Over the last 102 years, a lot of discussion was being held about the psychoanalytic conception of the "death drive," but still with inconclusive results. In this paper, we start with a brief review of Freud's conception, followed by a comprised overview of its subsequent support or criticisms. The core of our argument is a systematic review of current biochemical research about two proposed manifestations of the "death drive," which could hopefully move the discussion to the realm of science. It was already established that drive satisfaction leads to the secretion of beta-endorphins, and research evidence also shows that the same biochemical mechanisms get activated in the case of masochism and the gambling disorder but only if they are preceded by chronic frustration of the essential drives. We conclude that the actual situation is more complex than Freud hypothesized, and that a fundamental revision of the psychoanalytic drive theory is necessary.

摘要

在过去的102年里,人们对“死亡驱力”的精神分析概念进行了大量讨论,但结果仍无定论。在本文中,我们首先简要回顾弗洛伊德的概念,然后对其后续的支持或批评进行全面概述。我们论证的核心是对当前关于“死亡驱力”两种假定表现形式的生化研究进行系统回顾,这有望将讨论推进到科学领域。已经确定驱力满足会导致β-内啡肽的分泌,研究证据还表明,在受虐狂和赌博障碍的情况下,相同的生化机制会被激活,但前提是基本驱力长期受挫。我们得出结论,实际情况比弗洛伊德假设的更为复杂,精神分析驱力理论有必要进行根本性修订。

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