Wyeth N C
Med Phys. 1987 Jul-Aug;14(4):619-26. doi: 10.1118/1.596028.
Previous experiments have shown that murine ocular lenses immersed in saline in vitro suffered microscopic physical damage (including cataractogenic changes) after exposure to pulsed microwaves under conditions in which the temperature rise was negligible. The suspected coupling mechanism is thermoelastic transduction (TET). To test this hypothesis, laser interferometry was used to measure the lens surface motion induced by pulsed microwaves. Although experimental constraints delayed the recording of surface motion data until 0.5 ms after the microwave pulse, damped oscillatory motion was measured over an interval of several milliseconds. A theoretical model is presented to explain the observed motion as natural oscillation modes of an elastic sphere excited by TET waves produced in the surrounding saline. Effects of the lens capsule are also discussed. The conclusions reached are that the observed motion is TET induced, but the lens damage occurs during the passage of the initial excitatory TET waves through the lens, an event which could not be monitored by this experiment.
先前的实验表明，体外浸泡在盐水中的小鼠眼球晶状体，在温度上升可忽略不计的条件下暴露于脉冲微波后，会遭受微观物理损伤（包括致白内障变化）。疑似的耦合机制是热弹性转导（TET）。为了验证这一假设，使用激光干涉测量法来测量脉冲微波引起的晶状体表面运动。尽管实验限制使得表面运动数据的记录延迟到微波脉冲后0.5毫秒，但在几毫秒的时间间隔内测量到了阻尼振荡运动。本文提出了一个理论模型，将观察到的运动解释为由周围盐水中产生的TET波激发的弹性球体的自然振荡模式。还讨论了晶状体囊的影响。得出的结论是，观察到的运动是由TET诱导的，但晶状体损伤发生在初始兴奋性TET波穿过晶状体的过程中，而这一事件无法通过该实验进行监测。
