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CWF19L1相关疾病的表型和分子谱扩展

Expansion of the phenotypic and molecular spectrum of CWF19L1-related disorder.

作者信息

Alvarez Carolina, Grimmel Mona, Ebrahimi-Fakhari Darius, Paul Victoria G, Deininger Natalie, Riess Angelika, Haack Tobias, Gardella Elena, Møller Rikke S, Bayat Allan

机构信息

Department for genetics and personalized medicine, Danish Epilepsy Centre, Dianalund, Denmark.

Department of Pediatric Neurology, Avanced Epilepsy Center, Clínica Las Condes, Santiago, Chile.

出版信息

Clin Genet. 2023 May;103(5):566-573. doi: 10.1111/cge.14275. Epub 2023 Jan 11.

DOI:10.1111/cge.14275
PMID:36453471
Abstract

Pathogenic variants in CWF19L1 lead to a rare autosomal recessive form of hereditary ataxia with only seven cases reported to date. Here, we describe four additional unrelated patients with biallelic variants in CWF19L1 (age range: 6-22 years) and provide a comprehensive review of the literature. The clinical spectrum was broad, including mild to profound global developmental delay; global or motor regression in infancy or adolescence; childhood-onset ataxia and cerebellar atrophy; and early-onset epilepsy. Since only two previously reported patients were adults, our cohort expands our understanding of the evolution of symptoms from childhood into early adulthood. Taken together, we describe that CWF19L1-related disorder presents with developmental and epileptic encephalopathy with treatment-resistant seizures and intellectual disability in childhood followed by progressive ataxia and other extrapyramidal movement disorders in adolescence.

摘要

CWF19L1基因的致病性变异会导致一种罕见的常染色体隐性遗传性共济失调,迄今为止仅有7例病例报道。在此,我们描述了另外4例携带CWF19L1基因双等位基因变异的无亲缘关系患者(年龄范围:6至22岁),并对相关文献进行了全面综述。临床症状谱广泛,包括轻度至重度的全面发育迟缓;婴儿期或青春期的全面或运动功能倒退;儿童期起病的共济失调和小脑萎缩;以及早发性癫痫。由于之前报道的患者中只有2例是成年人,我们的队列扩展了我们对症状从儿童期到成年早期演变过程的理解。综合来看,我们描述了CWF19L1相关疾病表现为发育性和癫痫性脑病,儿童期有难治性癫痫发作和智力残疾症状,随后在青少年期出现进行性共济失调和其他锥体外系运动障碍。

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Expansion of the phenotypic and molecular spectrum of CWF19L1-related disorder.CWF19L1相关疾病的表型和分子谱扩展
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