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消除抗性:真菌效应蛋白靶向WAK可变剪接变体以实现毒力。

Disarm resistance: Fungal effectors target WAK alternative splicing variant for virulence.

作者信息

Liu Zunyong, Jian Yunqing, Shan Libo

机构信息

Department of Biochemistry and Biophysics, Texas A&M University, College Station, TX 77843, USA.

Department of Biochemistry and Biophysics, Texas A&M University, College Station, TX 77843, USA.

出版信息

Cell Rep. 2023 Jan 31;42(1):111939. doi: 10.1016/j.celrep.2022.111939. Epub 2023 Jan 4.

DOI:10.1016/j.celrep.2022.111939
PMID:36640313
Abstract

Molecular interactions between pathogen effectors and plant immunity underpin the arms race of disease resistance and susceptibility. In a recently published Cell Reports paper, Zuo et al. reported the mechanistic characterization of Fusarium graminearum CFEM effectors that dampen ZmWAK17-mediated defenses in maize (Zea mays).

摘要

病原体效应子与植物免疫之间的分子相互作用是抗病性和感病性军备竞赛的基础。在最近发表于《细胞报告》的一篇论文中,左等人报道了禾谷镰刀菌CFEM效应子的机制特征,这些效应子会削弱玉米(Zea mays)中ZmWAK17介导的防御。

相似文献

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Disarm resistance: Fungal effectors target WAK alternative splicing variant for virulence.消除抗性:真菌效应蛋白靶向WAK可变剪接变体以实现毒力。
Cell Rep. 2023 Jan 31;42(1):111939. doi: 10.1016/j.celrep.2022.111939. Epub 2023 Jan 4.
2
Fungal CFEM effectors negatively regulate a maize wall-associated kinase by interacting with its alternatively spliced variant to dampen resistance.真菌 CFEM 效应物通过与玉米壁相关激酶的可变剪接变体相互作用来负调控其活性,从而抑制抗性。
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