Finding Needles in the Gut Microbiota's Haystack.
作者信息
Aljadah Michael, Widlansky Michael E
机构信息
Division of Cardiovascular Medicine, Departments of Medicine and Pharmacology, Medical College of Wisconsin, Milwaukee.
出版信息
Circ Res. 2023 Jan 20;132(2):182-184. doi: 10.1161/CIRCRESAHA.122.322354. Epub 2023 Jan 19.
Abstract
摘要
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2
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Nat Commun. 2022 Jul 5;13(1):3863. doi: 10.1038/s41467-022-31502-1.
3
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Hypertension. 2020 Jul;76(1):101-112. doi: 10.1161/HYPERTENSIONAHA.120.14759. Epub 2020 Jun 10.
4
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7
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