Movement disorder associated with abnormal copper metabolism and decreased blood antioxidants.
作者信息
Pall H S, Williams A C, Blake D R, Winyard P, Chirico S, Brailsford S
出版信息
J Neurol Neurosurg Psychiatry. 1987 Sep;50(9):1234-5. doi: 10.1136/jnnp.50.9.1234.
Abstract
摘要
相似文献
1
Movement disorder associated with abnormal copper metabolism and decreased blood antioxidants.与铜代谢异常和血液抗氧化剂减少相关的运动障碍。
J Neurol Neurosurg Psychiatry. 1987 Sep;50(9):1234-5. doi: 10.1136/jnnp.50.9.1234.
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Metabolic pathways of red blood cell copper in normal humans and in Wilson's disease.
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本文引用的文献
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Degradation of deoxyribonucleic acid by a 1,10-phenanthroline-copper complex: the role of hydroxyl radicals.1,10-菲咯啉-铜配合物对脱氧核糖核酸的降解作用：羟基自由基的作用
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Copper-phenanthroline-induced site-specific oxygen-radical damage to DNA. Detection of loosely bound trace copper in biological fluids.铜-菲咯啉诱导的DNA位点特异性氧自由基损伤。生物体液中松散结合的痕量铜的检测。
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Concurrent exposure of lead and manganese to iron-deficient rats: effect on lipid peroxidation and contents of some metals in the brain.缺铁大鼠同时暴露于铅和锰：对脂质过氧化及大脑中某些金属含量的影响。
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Etiology of Parkinson's disease: A research strategy.
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A hereditary disorder with dementia, spastic dysarthria, vertical eye movement paresis, gait disturbance, splenomegaly, and abnormal copper metabolism.
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Late-onset Wilson's disease with neurological involvement in the absence of Kayser-Fleischer rings.无Kayser-Fleischer环的伴有神经受累的迟发性威尔逊病
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Coincidence of Wilson's disease with other movement disorders in the same family.威尔逊氏病与同一家族中的其他运动障碍并存。
J Neurol Neurosurg Psychiatry. 1986 Feb;49(2):221-2. doi: 10.1136/jnnp.49.2.221.
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A direct enzymic method for the determination of reduced glutathione in blood and other tissues.一种用于测定血液及其他组织中还原型谷胱甘肽的直接酶法。
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Free-radical oxidation and antioxidants.
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