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三例具有非典型眼底表现的急性视网膜坏死

THREE CASES OF ACUTE RETINAL NECROSIS WITH ATYPICAL FUNDUS FINDINGS.

作者信息

Chujo Shinichiro, Matsubara Hisashi, Ichio Atsushi, Matsui Yoshitsugu, Sugimoto Masahiko, Kondo Mineo

机构信息

Department of Ophthalmology, Mie University Graduate School of Medicine, Mie, Japan; and.

Sakuranomori Eye Clinic, Suzuka, Mie, Japan.

出版信息

Retin Cases Brief Rep. 2024 May 1;18(3):308-311. doi: 10.1097/ICB.0000000000001379. Epub 2024 Apr 18.

DOI:10.1097/ICB.0000000000001379
PMID:36730471
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11027986/
Abstract

PURPOSE

To determine the mechanism for the development of segmental granular lesions along the retinal vessels in eyes with acute retinal necrosis.

METHOD

This was a retrospective analysis of the medical records of three eyes of three patients who were diagnosed with acute retinal necrosis that had atypical segmental granular lesions aligned along the retinal vessels.

RESULTS

The segmental granular lesions were present on the retinal arteries and veins throughout the retina. Optical coherence tomography showed that the granular lesions protruded into the vitreous cavity. Histopathologic examinations confirmed that the lesions were made up of lymphocytes.

CONCLUSION

We suggest that the granular lesions were formed by a mechanism similar to that of HTLV-1-associated uveitis. We also found that the granular lesions disappeared soon after vitrectomy.

摘要

目的

确定急性视网膜坏死患者眼中沿视网膜血管出现节段性颗粒状病变的发生机制。

方法

对3例诊断为急性视网膜坏死且伴有沿视网膜血管排列的非典型节段性颗粒状病变的患者的3只眼的病历进行回顾性分析。

结果

整个视网膜的视网膜动脉和静脉均出现节段性颗粒状病变。光学相干断层扫描显示颗粒状病变突入玻璃体腔。组织病理学检查证实病变由淋巴细胞组成。

结论

我们认为颗粒状病变是由与人类嗜T淋巴细胞病毒1型相关葡萄膜炎相似的机制形成的。我们还发现玻璃体切除术后颗粒状病变很快消失。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/8c9157e3026c/cabr-18-308-g008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/5bc7885e4782/cabr-18-308-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/f49945280c98/cabr-18-308-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/b679eb7f01a4/cabr-18-308-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/182bff7ebeb6/cabr-18-308-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/c208c9f0241c/cabr-18-308-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/c231f8b31f6c/cabr-18-308-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/3d4488116ec6/cabr-18-308-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/8c9157e3026c/cabr-18-308-g008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/5bc7885e4782/cabr-18-308-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/f49945280c98/cabr-18-308-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/b679eb7f01a4/cabr-18-308-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/182bff7ebeb6/cabr-18-308-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/c208c9f0241c/cabr-18-308-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/c231f8b31f6c/cabr-18-308-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/3d4488116ec6/cabr-18-308-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c94b/11027986/8c9157e3026c/cabr-18-308-g008.jpg

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