Kryzhanovskiĭ G N, Lobasiuk B A, Mokhovikov A N, Bartsevich L B, Mosketi K V
Biull Eksp Biol Med. 1987 Oct;104(10):405-8.
The experiments on adult freely behaving cats have shown that the injection of penicillin into the internal part (TI) of bed nucleus of stria terminalis (BST) caused the formation of the generator of pathologically enhanced excitation (GPEE). This part of BST became hyperactive and played the role of a pathologic determinant which caused the formation of a complex pathologic system and behavioural disorders with catatonia, stereotypy, food rejection, eating of uneatable things, hypoalgesia, zoosocial behavioural changes, depression of different emotions, etc. The development of these disorders was progressing and was accompanied by corresponding electrophysiological activity in TI and other brain structures studied. Coagulation of GPEE zone at the early stages of the process prevented the development of the above disorders.
对成年自由活动猫的实验表明，向终纹床核（BST）的内侧部（TI）注射青霉素会导致病理性增强兴奋发生器（GPEE）的形成。BST的这一部分变得过度活跃，并起到病理决定因素的作用，导致形成复杂的病理系统和行为障碍，包括紧张症、刻板行为、拒食、吃不能吃的东西、痛觉减退、社会行为改变、不同情绪的抑制等。这些障碍不断发展，并伴有TI及所研究的其他脑结构中的相应电生理活动。在此过程的早期阶段对GPEE区域进行凝固可阻止上述障碍的发展。
