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Apelin is altered in subjects with autosomal dominant polycystic kidney disease and preserved kidney function.

作者信息

Janssens Peter, Cools Wilfried, de Mota Nadia, Decuypere Jean-Paul, Torres Vicente, Wissing Karl Martin, Vennekens Rudi, Bammens Bert, Llorens-Cortes Catherine, Mekahli Djalila

机构信息

Department of Nephrology and Arterial Hypertension, Universitair Ziekenhuis Brussel (UZ Brussel), Vrije Universiteit Brussel, Brussels, Belgium.

PKD research group, Department of Cellular and Molecular Medicine, KU Leuven, Leuven, Belgium.

出版信息

Nephrol Dial Transplant. 2023 Jul 31;38(8):1907-1911. doi: 10.1093/ndt/gfad056.

DOI:10.1093/ndt/gfad056
PMID:36945109
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10387394/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d7c7/10387394/b142cde0d4e4/gfad056fig1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d7c7/10387394/b142cde0d4e4/gfad056fig1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d7c7/10387394/b142cde0d4e4/gfad056fig1.jpg

相似文献

1
Apelin is altered in subjects with autosomal dominant polycystic kidney disease and preserved kidney function.阿片肽在常染色体显性遗传性多囊肾病且肾功能尚好的患者中发生改变。
Nephrol Dial Transplant. 2023 Jul 31;38(8):1907-1911. doi: 10.1093/ndt/gfad056.
2
Apelin and copeptin: two opposite biomarkers associated with kidney function decline and cyst growth in autosomal dominant polycystic kidney disease.Apelin 和 copeptin:两种与常染色体显性多囊肾病肾功能下降和囊肿生长相关的相反生物标志物。
Peptides. 2013 Nov;49:1-8. doi: 10.1016/j.peptides.2013.08.007. Epub 2013 Aug 21.
3
Evaluation of Fibrosis Markers: Apelin and Transforming Growth Factor-β1 in Autosomal Dominant Polycystic Kidney Disease Patients.常染色体显性多囊肾病患者纤维化标志物Apelin和转化生长因子-β1的评估
Ther Apher Dial. 2016 Oct;20(5):517-522. doi: 10.1111/1744-9987.12412. Epub 2016 Mar 17.
4
Autosomal recessive polycystic kidney disease does not map to the second gene locus for autosomal dominant polycystic kidney disease on chromosome 4.常染色体隐性多囊肾病并不定位于4号染色体上常染色体显性多囊肾病的第二个基因位点。
Hum Genet. 1994 Jun;93(6):697-8. doi: 10.1007/BF00201574.
5
Autosomal dominant polycystic kidney disease.
Am J Kidney Dis. 2006 Jun;47(6):A37, e73-5.
6
Automated Segmentation of Kidneys from MR Images in Patients with Autosomal Dominant Polycystic Kidney Disease.常染色体显性多囊肾病患者肾脏的磁共振图像自动分割
Clin J Am Soc Nephrol. 2016 Apr 7;11(4):576-84. doi: 10.2215/CJN.08300815. Epub 2016 Jan 21.
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Newly diagnosed polycystic kidney disease: what to do with the family?
Aust N Z J Med. 1995 Oct;25(5):469-71. doi: 10.1111/j.1445-5994.1995.tb01489.x.
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Current advances in molecular genetics of autosomal-dominant polycystic kidney disease.常染色体显性多囊肾病分子遗传学的当前进展
Curr Opin Nephrol Hypertens. 2001 Jan;10(1):23-31. doi: 10.1097/00041552-200101000-00005.
9
Progress in molecular genetics of autosomal dominant polycystic kidney disease.
Front Biosci. 2000 Mar 1;5:D312-20. doi: 10.2741/trudel.
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Coronary flow velocity reserve and carotid intima media thickness in patients with autosomal dominant polycystic kidney disease: from impaired tubules to impaired carotid and coronary arteries.常染色体显性多囊肾病患者的冠状动脉血流储备和颈动脉内膜中层厚度:从肾小管受损到颈动脉和冠状动脉受损
Clin J Am Soc Nephrol. 2008 Jul;3(4):986-91. doi: 10.2215/CJN.02330607. Epub 2008 Mar 19.

本文引用的文献

1
On Methods for the Measurement of the Apelin Receptor Ligand Apelin.关于阿皮林受体配体阿皮林测量方法的研究
Sci Rep. 2022 May 11;12(1):7763. doi: 10.1038/s41598-022-11835-z.
2
Vaptans or voluntary increased hydration to protect the kidney: how do they compare?缬沙坦或自愿增加水合作用以保护肾脏:它们如何比较?
Nephrol Dial Transplant. 2023 Feb 28;38(3):562-574. doi: 10.1093/ndt/gfab278.
3
The therapeutic potential of apelin in kidney disease.阿皮林在肾脏疾病中的治疗潜力。
Nat Rev Nephrol. 2021 Dec;17(12):840-853. doi: 10.1038/s41581-021-00461-z. Epub 2021 Aug 13.
4
The emerging role of the apelinergic system in kidney physiology and disease.阿片肽系统在肾脏生理学和疾病中的新作用。
Nephrol Dial Transplant. 2022 Nov 23;37(12):2314-2326. doi: 10.1093/ndt/gfab070.
5
A metabolically stable apelin-17 analog decreases AVP-induced antidiuresis and improves hyponatremia.一种代谢稳定的阿肽-17 类似物可减少 AVP 诱导的抗利尿作用并改善低钠血症。
Nat Commun. 2021 Jan 12;12(1):305. doi: 10.1038/s41467-020-20560-y.
6
Autosomal dominant polycystic kidney disease.常染色体显性遗传性多囊肾病。
Lancet. 2019 Mar 2;393(10174):919-935. doi: 10.1016/S0140-6736(18)32782-X. Epub 2019 Feb 25.
7
Development of original metabolically stable apelin-17 analogs with diuretic and cardiovascular effects.具有利尿和心血管效应的新型代谢稳定的apelin-17类似物的研发。
FASEB J. 2017 Feb;31(2):687-700. doi: 10.1096/fj.201600784R. Epub 2016 Nov 4.
8
Apela Regulates Fluid Homeostasis by Binding to the APJ Receptor to Activate Gi Signaling.Apela通过与APJ受体结合以激活Gi信号传导来调节液体稳态。
J Biol Chem. 2015 Jul 24;290(30):18261-8. doi: 10.1074/jbc.M115.648238. Epub 2015 May 20.
9
Apelin and copeptin: two opposite biomarkers associated with kidney function decline and cyst growth in autosomal dominant polycystic kidney disease.Apelin 和 copeptin:两种与常染色体显性多囊肾病肾功能下降和囊肿生长相关的相反生物标志物。
Peptides. 2013 Nov;49:1-8. doi: 10.1016/j.peptides.2013.08.007. Epub 2013 Aug 21.
10
Relationship of copeptin, a surrogate marker for arginine vasopressin, with change in total kidney volume and GFR decline in autosomal dominant polycystic kidney disease: results from the CRISP cohort. copeptin,精氨酸加压素的替代标志物,与常染色体显性多囊肾病患者的总肾体积变化和 GFR 下降的关系:来自 CRISP 队列的结果。
Am J Kidney Dis. 2013 Mar;61(3):420-9. doi: 10.1053/j.ajkd.2012.08.038. Epub 2012 Oct 22.