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病毒入侵时利用宿主短线性基序介导相互作用的分子模拟。

Molecular mimicry of host short linear motif-mediated interactions utilised by viruses for entry.

机构信息

School of Bioscience, Indian Institute of Technology Kharagpur, Kharagpur, West Bengal, 721302, India.

Department of Biochemistry, Albert Einstein College of Medicine, Bronx, NY, 10461, USA.

出版信息

Mol Biol Rep. 2023 May;50(5):4665-4673. doi: 10.1007/s11033-023-08389-2. Epub 2023 Apr 4.

Abstract

Viruses are obligate intracellular parasites that depend on host cellular machinery for performing even basic biological functions. One of the many ways they achieve this is through molecular mimicry, wherein the virus mimics a host sequence or structure, thereby being able to hijack the host's physiological interactions for its pathogenesis. Such adaptations are specific recognitions that often confer tissue and species-specific tropisms to the virus, and enable the virus to utilise previously existing host signalling networks, which ultimately aid in further steps of viral infection, such as entry, immune evasion and spread. A common form of sequence mimicry utilises short linear motifs (SLiMs). SLiMs are short-peptide sequences that mediate transient interactions and are major elements in host protein interaction networks. This work is aimed at providing a comprehensive review of current literature of some well-characterised SLiMs that play a role in the attachment and entry of viruses into host cells, which mimic physiological receptor-ligand interactions already present in the host. Considering recent trends in emerging diseases, further research on such motifs involved in viral entry can help in the discovery of previously unknown cellular receptors utilised by viruses, as well as help in the designing of targeted therapeutics such as vaccines or inhibitors directed towards these interactions.

摘要

病毒是专性细胞内寄生虫,甚至要依赖宿主细胞机制来执行基本的生物学功能。它们实现这一点的方法之一是通过分子模拟,即病毒模拟宿主序列或结构,从而能够劫持宿主的生理相互作用来实现其发病机制。这种适应性是特定的识别,通常赋予病毒组织和物种特异性趋向性,并使病毒能够利用宿主先前存在的信号网络,这最终有助于病毒感染的进一步步骤,如进入、免疫逃逸和传播。序列模拟的一种常见形式是利用短线性基序(SLiMs)。SLiMs 是介导短暂相互作用的短肽序列,是宿主蛋白相互作用网络的主要元件。这项工作旨在对一些已充分研究的 SLiMs 的文献进行全面综述,这些 SLiMs 在病毒进入宿主细胞的附着和进入中发挥作用,模拟宿主中已经存在的生理受体-配体相互作用。考虑到新发疾病的最新趋势,对病毒进入过程中涉及的此类基序的进一步研究,有助于发现病毒利用的先前未知的细胞受体,并有助于设计针对这些相互作用的靶向治疗药物,如疫苗或抑制剂。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c224/10072811/a219c39d30f2/11033_2023_8389_Fig1_HTML.jpg

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