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Mechanical and matrix effects of short and long-duration exposure to beta-aminopropionitrile in elastase-induced model abdominal aortic aneurysm in mice.在弹性蛋白酶诱导的小鼠腹主动脉瘤模型中，短期和长期暴露于β-氨基丙腈的机械和基质效应。
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2
Translating mouse models of abdominal aortic aneurysm to the translational needs of vascular surgery.将腹主动脉瘤小鼠模型转化为血管外科的转化需求。
JVS Vasc Sci. 2021 Mar 3;2:219-234. doi: 10.1016/j.jvssci.2021.01.002. eCollection 2021.
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Biomechanical consequences of compromised elastic fiber integrity and matrix cross-linking on abdominal aortic aneurysmal enlargement.弹性纤维完整性和基质交联受损对腹主动脉瘤扩大的生物力学后果。
Acta Biomater. 2021 Oct 15;134:422-434. doi: 10.1016/j.actbio.2021.07.059. Epub 2021 Jul 29.
4
High-frequency murine ultrasound provides enhanced metrics of BAPN-induced AAA growth.高频鼠超声提供增强的 BAPN 诱导的 AAA 生长的指标。
Am J Physiol Heart Circ Physiol. 2019 Nov 1;317(5):H981-H990. doi: 10.1152/ajpheart.00300.2019. Epub 2019 Sep 27.
5
A novel chronic advanced stage abdominal aortic aneurysm murine model.一种新型慢性晚期腹主动脉瘤小鼠模型。
J Vasc Surg. 2017 Jul;66(1):232-242.e4. doi: 10.1016/j.jvs.2016.07.105. Epub 2017 Mar 6.
6
Development of a novel murine model of aortic aneurysms using peri-adventitial elastase.使用血管外膜弹性蛋白酶建立新型小鼠主动脉瘤模型
Surgery. 2012 Aug;152(2):238-46. doi: 10.1016/j.surg.2012.02.010.
7
Matrix metalloproteinases 2 and 9 work in concert to produce aortic aneurysms.基质金属蛋白酶2和9协同作用导致主动脉瘤。
J Clin Invest. 2002 Sep;110(5):625-32. doi: 10.1172/JCI15334.
8
Targeted gene disruption of matrix metalloproteinase-9 (gelatinase B) suppresses development of experimental abdominal aortic aneurysms.基质金属蛋白酶-9（明胶酶B）的靶向基因破坏抑制实验性腹主动脉瘤的发展。
J Clin Invest. 2000 Jun;105(11):1641-9. doi: 10.1172/JCI8931.
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Angiotensin II promotes atherosclerotic lesions and aneurysms in apolipoprotein E-deficient mice.血管紧张素II可促进载脂蛋白E缺乏小鼠的动脉粥样硬化病变和动脉瘤形成。
J Clin Invest. 2000 Jun;105(11):1605-12. doi: 10.1172/JCI7818.

Abdominal aortic aneurysms: insights into mechanical and extracellular matrix effects from mouse models.

作者信息

Lu Hong S, Sawada Hisashi, Daugherty Alan

机构信息

Saha Cardiovascular Research Center, Department of Physiology, University of Kentucky, Lexington, KY.

出版信息

JVS Vasc Sci. 2023 Feb 18;4:100099. doi: 10.1016/j.jvssci.2023.100099. eCollection 2023.

DOI:10.1016/j.jvssci.2023.100099
PMID:37180337
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10172769/
Abstract
摘要