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Intracellular effects of myocardial ischaemia and reperfusion: role of calcium and oxygen.

作者信息

Ferrari R, Ceconi C, Curello S, Cargnoni A, Agnoletti G, Boffa G M, Visioli O

出版信息

Eur Heart J. 1986 May;7 Suppl A:3-12.

PMID:3720774
Abstract
摘要

相似文献

1
Intracellular effects of myocardial ischaemia and reperfusion: role of calcium and oxygen.心肌缺血与再灌注的细胞内效应：钙与氧的作用
Eur Heart J. 1986 May;7 Suppl A:3-12.
2
[Comparative protective effect of gallopamil in myocardial ischemia and reperfusion].
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Oxygen-mediated myocardial damage during ischaemia and reperfusion: role of the cellular defences against oxygen toxicity.缺血再灌注期间氧介导的心肌损伤：细胞抗氧毒性防御机制的作用
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[Energy metabolism and cell injury in ischemic heart].
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Role of oxygen in myocardial ischaemic and reperfusion damage: effect of alpha-tocopherol.
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Metabolic processes leading to myocardial cell death.
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[Pathophysiological causal-factors of myocardial necrosis and infarct].[心肌坏死与梗死的病理生理因果因素]
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[Protection of oxyphenamone against cardiac arrest-reperfusion injury of isolated rat hearts].[奥昔苯酮对离体大鼠心脏骤停-再灌注损伤的保护作用]
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Metabolic changes during post-ischaemic reperfusion.
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引用本文的文献

1
Structural and Functional Remodeling of Mitochondria in Cardiac Diseases.心脏疾病中线粒体的结构和功能重塑。
Int J Mol Sci. 2021 Apr 17;22(8):4167. doi: 10.3390/ijms22084167.
2
Ischemia-reperfusion leads to depletion of glutathione content and augmentation of malondialdehyde production in the rat heart from overproduction of oxidants: can caffeic acid phenethyl ester (CAPE) protect the heart?缺血再灌注会导致大鼠心脏中谷胱甘肽含量减少以及丙二醛生成增加，这是由于氧化剂产生过多所致：咖啡酸苯乙酯（CAPE）能否保护心脏？
Mol Cell Biochem. 2005 May;273(1-2):169-75. doi: 10.1007/s11010-005-0551-8.
3
Effect of lacidipine on ischaemic and reperfused isolated rabbit hearts.
拉西地平对离体兔缺血再灌注心脏的影响。
Mol Cell Biochem. 1993 Aug 11;125(1):73-86. doi: 10.1007/BF00926837.
4
Mitochondrial energy production and cation control in myocardial ischaemia and reperfusion.心肌缺血再灌注中的线粒体能量产生与阳离子调控
Basic Res Cardiol. 1993 Sep-Oct;88(5):495-512. doi: 10.1007/BF00795415.
5
Calcium antagonists and acute myocardial ischemia: comparative effects of gallopamil and nifedipine on ischemia-induced and reperfusion-induced ventricular arrhythmias, epicardial conduction times, and ventricular fibrillation thresholds.钙拮抗剂与急性心肌缺血：加洛帕米与硝苯地平对缺血诱导及再灌注诱导的室性心律失常、心外膜传导时间和室颤阈值的比较作用
Cardiovasc Drugs Ther. 1987 Dec;1(4):367-76. doi: 10.1007/BF02209078.
6
Mitochondrial calcium in hearts subjected to lipid peroxidation with contracture development.
Basic Res Cardiol. 1989 Nov-Dec;84(6):646-52. doi: 10.1007/BF01906949.
7
Effects of iloprost (ZK 36374) on glutathione status during ischaemia and reperfusion of rabbit isolated hearts.伊洛前列素（ZK 36374）对兔离体心脏缺血再灌注期间谷胱甘肽状态的影响。
Br J Pharmacol. 1989 Oct;98(2):678-84. doi: 10.1111/j.1476-5381.1989.tb12643.x.
8
The occurrence of oxidative stress during reperfusion in experimental animals and men.
Cardiovasc Drugs Ther. 1991 Mar;5 Suppl 2:277-87. doi: 10.1007/BF00054749.
9
Stunning: a radical re-view.顿抑：一次彻底的重新审视。
Cardiovasc Drugs Ther. 1991 Oct;5(5):853-76. doi: 10.1007/BF00053547.
10
Protective effects of calcium antagonists against ischaemia and reperfusion damage.钙拮抗剂对缺血和再灌注损伤的保护作用。
Drugs. 1991;42 Suppl 1:14-26; discussion 26-7. doi: 10.2165/00003495-199100421-00005.