Does multiple sclerosis have a zoonotic origin? Correlations with lymphocytic choriomeningitis virus infection.
作者信息
Hogeboom Charissa
机构信息
Independent, Albany, CA, United States.
出版信息
Front Immunol. 2023 Jun 16;14:1217176. doi: 10.3389/fimmu.2023.1217176. eCollection 2023.
Abstract
摘要
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Theiler's virus-induced demyelinating disease as an infectious model of progressive multiple sclerosis.泰勒氏病毒诱导的脱髓鞘疾病作为进行性多发性硬化症的感染模型。
Front Mol Neurosci. 2022 Oct 13;15:1019799. doi: 10.3389/fnmol.2022.1019799. eCollection 2022.
2
Longitudinal analysis reveals high prevalence of Epstein-Barr virus associated with multiple sclerosis.纵向分析显示，多发性硬化症与 Epstein-Barr 病毒的高患病率相关。
Science. 2022 Jan 21;375(6578):296-301. doi: 10.1126/science.abj8222. Epub 2022 Jan 13.
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Lymphocytic choriomeningitis virus infection of dendritic cells interferes with TLR-induced IL-12/IL-23 cytokine production in an IL-10 independent manner.淋巴细胞性脉络丛脑膜炎病毒感染树突状细胞以不依赖于白细胞介素-10 的方式干扰 TLR 诱导的白细胞介素-12/白细胞介素-23 细胞因子的产生。
Cytokine. 2018 Aug;108:105-114. doi: 10.1016/j.cyto.2018.03.017. Epub 2018 Mar 27.
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Epstein-Barr virus is present in the brain of most cases of multiple sclerosis and may engage more than just B cells.爱泼斯坦-巴尔病毒存在于大多数多发性硬化症病例的大脑中，并且可能不仅仅影响B细胞。
PLoS One. 2018 Feb 2;13(2):e0192109. doi: 10.1371/journal.pone.0192109. eCollection 2018.
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The innovative development in interferon beta treatments of relapsing-remitting multiple sclerosis.干扰素 β 治疗复发缓解型多发性硬化症的创新发展。
Brain Behav. 2017 May 8;7(6):e00696. doi: 10.1002/brb3.696. eCollection 2017 Jun.
6
Defective T-cell control of Epstein-Barr virus infection in multiple sclerosis.多发性硬化症中爱泼斯坦-巴尔病毒感染的T细胞控制缺陷
Clin Transl Immunology. 2017 Jan 20;6(1):e126. doi: 10.1038/cti.2016.87. eCollection 2017 Jan.
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Myeloid dendritic cells exhibit defects in activation and function in patients with multiple sclerosis.在多发性硬化症患者中，髓样树突状细胞在激活和功能方面存在缺陷。
J Neuroimmunol. 2016 Dec 15;301:53-60. doi: 10.1016/j.jneuroim.2016.10.007. Epub 2016 Nov 2.
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Peptide motif analysis predicts lymphocytic choriomeningitis virus as trigger for multiple sclerosis.肽基序分析预测淋巴细胞性脉络丛脑膜炎病毒是多发性硬化症的触发因素。
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Endogenous interferon-β-inducible gene expression and interferon-β-treatment are associated with reduced T cell responses to myelin basic protein in multiple sclerosis.内源性干扰素-β诱导基因表达和干扰素-β治疗与多发性硬化症中T细胞对髓鞘碱性蛋白的反应降低有关。
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