Wright K C, Agre J C, Wilson B C, Theologides A
Arch Phys Med Rehabil. 1986 Aug;67(8):566-7.
Autonomic dysreflexia and catecholamine secreting tumor, each of which causes paroxysmal hypertension, coexisted in a young man. Two years after neuroblastoma was diagnosed, he developed T4 incomplete paraplegia due to metastases to the spine at T5 and L3 levels. Shortly after the onset of paraplegia, paroxysmal hypertension developed. The hypertension was controlled adequately by good bowel and bladder management and oral clonidine. The paroxysmal hypertension is believed to have resulted from the synergistic effect of the high levels of circulating catecholamines from the tumor and the disruption of autonomic pathways.
一名年轻男性同时患有自主神经反射异常和分泌儿茶酚胺的肿瘤，二者均导致阵发性高血压。神经母细胞瘤确诊两年后，他因T5和L3水平的脊柱转移而出现T4不完全性截瘫。截瘫发作后不久，出现了阵发性高血压。通过良好的肠道和膀胱管理以及口服可乐定，高血压得到了充分控制。据信，阵发性高血压是由肿瘤释放的高水平循环儿茶酚胺与自主神经通路中断的协同作用所致。
