Moreira Filho P F, Freitas M R
Arq Neuropsiquiatr. 1986 Mar;44(1):67-72. doi: 10.1590/s0004-282x1986000100009.
A case of a young man with myoclonus in his right side after cerebral hypoxia is reported. This patient had cerebral hypoxia caused by injury in his left common carotid artery. After a few hours he had generalized convulsive seizures of tonic-clonic type and also a septic shock. As consciousness was regained, he developed action and intention myoclonus in his right-side. The EEG showed diffuse typical myoclonus potentials. Clonazepan 8 mg daily was used with good results. This is the first reference in the medical literature of the unilateral localization in the Lance-Adams syndrome. The authors think that in this case two abnormalities contributed to the cerebral anoxia: the common carotid artery injury and the septic shock.
报告了一例青年男性在脑缺氧后出现右侧肌阵挛的病例。该患者因左侧颈总动脉损伤导致脑缺氧。数小时后，他出现了全身性强直阵挛性惊厥以及感染性休克。意识恢复后，他右侧出现了动作性和意向性肌阵挛。脑电图显示弥漫性典型肌阵挛电位。每日使用8毫克氯硝西泮，效果良好。这是医学文献中关于兰斯-亚当斯综合征单侧定位的首次参考。作者认为在该病例中，有两个异常因素导致了脑缺氧：颈总动脉损伤和感染性休克。
