撤回：急性心肌缺血后，氧化损伤的线粒体以自噬外泌体方式激活GABARAPL1诱导的NLRP3炎性小体。
Retracted: Oxidative-Damaged Mitochondria Activate GABARAPL1-Induced NLRP3 Inflammasomes in an Autophagic-Exosome Manner after Acute Myocardial Ischemia.
作者信息
Longevity Oxidative Medicine And Cellular
出版信息
Oxid Med Cell Longev. 2023 Aug 2;2023:9824094. doi: 10.1155/2023/9824094. eCollection 2023.
Abstract
[This retracts the article DOI: 10.1155/2022/7958542.].
摘要
[本文撤回文章DOI: 10.1155/2022/7958542。]
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Retracted: Oxidative-Damaged Mitochondria Activate GABARAPL1-Induced NLRP3 Inflammasomes in an Autophagic-Exosome Manner after Acute Myocardial Ischemia.撤回：急性心肌缺血后，氧化损伤的线粒体以自噬外泌体方式激活GABARAPL1诱导的NLRP3炎性小体。
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本文引用的文献
1
Oxidative-Damaged Mitochondria Activate GABARAPL1-Induced NLRP3 Inflammasomes in an Autophagic-Exosome Manner after Acute Myocardial Ischemia.氧化损伤的线粒体通过自噬小体-外泌体途径激活 GABARAPL1 诱导的 NLRP3 炎性小体后发生急性心肌缺血。
Oxid Med Cell Longev. 2022 Sep 30;2022:7958542. doi: 10.1155/2022/7958542. eCollection 2022.
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