Vital C, Dumas P, Latinville D, Dib M, Vital A, Brechenmacher C
Acta Neuropathol. 1986;71(1-2):94-9. doi: 10.1007/BF00687968.
Inflammatory demyelinating polyradiculoneuropathies exhibit well-known ultrastructural lesions of the peripheral nerve, both in acute cases, i.e., Guillain-Barré syndrome, and in relapsing, sub-acute and chronic cases. We present a case of relapsing inflammatory demyelinating polyradiculoneuropathy in a diabetic patient with a biopsy exhibiting these lesions, as well as a widening of the outermost myelin lamellae in some fibers. Such associated lesions are classic in experimental inflammatory demyelinating polyradiculoneuropathies, but have not been reported in human pathology.
炎症性脱髓鞘性多发性神经根神经病在急性病例(即吉兰-巴雷综合征)以及复发型、亚急性和慢性病例中均表现出外周神经众所周知的超微结构病变。我们报告了一例糖尿病患者复发性炎症性脱髓鞘性多发性神经根神经病,其活检显示出这些病变,以及部分神经纤维最外层髓鞘板增宽。此类相关病变在实验性炎症性脱髓鞘性多发性神经根神经病中很典型,但在人体病理学中尚未见报道。