Tanaka S, Tanabe M
Acta Otolaryngol. 1986 Sep-Oct;102(3-4):315-24. doi: 10.3109/00016488609108682.
Living canine larynx was insufflated from the transected trachea with and without contractions of the lateral cricoarytenoid, cricothyroid and thyroarytenoid muscles, in order to study the control mechanism of vocal intensity at the glottal level. The contractions of the lateral cricoarytenoid and cricothyroid muscles were simulated with mechanical retraction, while the thyroarytenoid muscle was contracted with electrical stimulation. The vocal intensity (SPL), subglottal pressure and mean flow rate were measured simultaneously, and the aerodynamic power (pressure multiplied by flow rate) and the glottal resistance (pressure divided by flow rate) were calculated. At the same aerodynamic power, both the vocal intensity and resistance were increased by cordal adduction with contraction of the lateral cricoarytenoid muscle, whereas the vocal intensity was kept constant with contraction of the thyroarytenoid muscle in spite of increasing the resistance. Contraction of the cricothyroid muscle was inferred to influence the vocal intensity indirectly by altering the cordal adduction.
为了研究声门水平声音强度的控制机制，在切断气管的情况下，对活体犬的喉部进行充气，同时观察环杓侧肌、环甲肌和甲杓肌有无收缩。用机械牵拉模拟环杓侧肌和环甲肌的收缩，用电刺激使甲杓肌收缩。同时测量声音强度（声压级）、声门下压力和平均流速，并计算气动功率（压力乘以流速）和声门阻力（压力除以流速）。在相同的气动功率下，环杓侧肌收缩使声带内收，声音强度和阻力均增加；而甲杓肌收缩时，尽管阻力增加，但声音强度保持不变。推测环甲肌的收缩通过改变声带内收间接影响声音强度。
