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What Is Ulcerative Colitis?

作者信息

Voelker Rebecca

机构信息

Contributing Writer, JAMA.

出版信息

JAMA. 2024 Feb 27;331(8):716. doi: 10.1001/jama.2023.23814.

DOI:10.1001/jama.2023.23814
PMID:38306113
Abstract
摘要

相似文献

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What Is Ulcerative Colitis?什么是溃疡性结肠炎?
JAMA. 2024 Feb 27;331(8):716. doi: 10.1001/jama.2023.23814.
2
JAMA patient page. Ulcerative colitis.《美国医学会杂志》患者专页。溃疡性结肠炎
JAMA. 2012 Jan 4;307(1):104. doi: 10.1001/jama.2011.1889.
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Performance of tacrolimus in hospitalized patients with steroid-refractory acute severe ulcerative colitis.他克莫司在住院的类固醇难治性急性重度溃疡性结肠炎患者中的疗效。
World J Gastroenterol. 2019 Apr 7;25(13):1603-1617. doi: 10.3748/wjg.v25.i13.1603.
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Ulcerative Colitis: Making the Diagnosis.溃疡性结肠炎:诊断要点。
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Use of Novel Biological Agent in Severe Ulcerative Colitis with Poor Response to Initial Therapy: A Case Report.新型生物制剂在初始治疗反应不佳的重度溃疡性结肠炎中的应用:一例报告。
JNMA J Nepal Med Assoc. 2022 Sep 1;60(253):825-827. doi: 10.31729/jnma.7590.
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[Remission of ulcerative colitis after appendectomy: a case report].[阑尾切除术后溃疡性结肠炎缓解:一例报告]
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Review article: medical treatment of severe ulcerative colitis.综述文章:重度溃疡性结肠炎的医学治疗
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[Bronchiectasia revealing ulcerative colitis].支气管扩张症揭示溃疡性结肠炎
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Ulcerative colitis.溃疡性结肠炎
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Case of lacrimal gland inflammation associated with ulcerative colitis.泪腺炎合并溃疡性结肠炎病例。
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引用本文的文献

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Ulcerative Colitis: Advances in Pathogenesis, Biomarkers, and Therapeutic Strategies.溃疡性结肠炎:发病机制、生物标志物及治疗策略的进展
Pharmgenomics Pers Med. 2025 Sep 5;18:219-238. doi: 10.2147/PGPM.S536459. eCollection 2025.
2
Advances in the therapeutic applications of dichloroacetate as a metabolic regulator: A review.二氯乙酸作为代谢调节剂的治疗应用进展:综述
Medicine (Baltimore). 2025 Sep 5;104(36):e44295. doi: 10.1097/MD.0000000000044295.
3
Lithocholic acid ameliorates ulcerative colitis via the PXR/TLR4/NF-κB/NLRP3 signaling pathway and gut microbiota modulation.
石胆酸通过PXR/TLR4/NF-κB/NLRP3信号通路和肠道微生物群调节改善溃疡性结肠炎。
Cell Mol Life Sci. 2025 Sep 4;82(1):336. doi: 10.1007/s00018-025-05834-2.
4
Epigenomic Profiling Positions ATF7 as a Core Regulator of Colonic Inflammation.表观基因组分析确定ATF7为结肠炎症的核心调节因子。
J Cell Mol Med. 2025 Sep;29(17):e70831. doi: 10.1111/jcmm.70831.
5
A Comprehensive Review of Food-Derived Compounds Targeting Pyroptosis for Colitis Therapy: From Effects to Mechanisms.靶向焦亡用于结肠炎治疗的食物衍生化合物综述:从作用到机制
J Inflamm Res. 2025 Aug 27;18:11667-11688. doi: 10.2147/JIR.S531820. eCollection 2025.
6
Preclinical evidence for luteolin in ulcerative colitis: a meta-analysis and systematic review.木犀草素治疗溃疡性结肠炎的临床前证据:一项荟萃分析与系统评价
Front Pharmacol. 2025 Jul 30;16:1639644. doi: 10.3389/fphar.2025.1639644. eCollection 2025.
7
β-Sitosterol improves murine ulcerative colitis by inhibiting the expression of ribosomal proteins and the attempted polarization of type 1 macrophages.β-谷甾醇通过抑制核糖体蛋白的表达和1型巨噬细胞的尝试极化来改善小鼠溃疡性结肠炎。
Int J Colorectal Dis. 2025 Aug 5;40(1):169. doi: 10.1007/s00384-025-04974-y.
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Identification of differentially expressed genes associated with ferroptosis in ulcerative colitis.溃疡性结肠炎中与铁死亡相关的差异表达基因的鉴定
PLoS One. 2025 Jul 29;20(7):e0327990. doi: 10.1371/journal.pone.0327990. eCollection 2025.
9
TLR4/NF-κB-mediated M1 macrophage polarization contributes to the promotive effects of ETS2 on ulcerative colitis.TLR4/NF-κB介导的M1巨噬细胞极化有助于ETS2对溃疡性结肠炎的促进作用。
Eur J Med Res. 2025 Jul 24;30(1):668. doi: 10.1186/s40001-025-02947-z.
10
The deficiency of chymase mast cell protease 4 exacerbates dextran sulfate sodium salt-induced colitis in mice and is associated with altered microbiota and metabolome profiles.糜酶肥大细胞蛋白酶4缺乏会加重葡聚糖硫酸钠诱导的小鼠结肠炎,并与微生物群和代谢组谱的改变有关。
Front Cell Infect Microbiol. 2025 Jul 8;15:1481927. doi: 10.3389/fcimb.2025.1481927. eCollection 2025.