Albouy R P
Doc Ophthalmol. 1985 Aug 15;60(1):45-69. doi: 10.1007/BF00164569.
The use of prisms in treatment of strabismus not only varies according to the author involved but often, a same author will change his opinion on the subject as time goes by. In the first case aetiopathological conceptions and the finality of treatment differ, in the second case improved theoretical knowledge and new possibilities in examination techniques and treatment etc., come into play. For this latter reason we wish to give complementary information and generally revise the publication on the use of prisms published in 1973 (18). The revised version in no way invalidates the previous work, it brings it up to date and gives further information on the severe forms of strabismus which, at the time of the first publication, constituted the failure group. These severe forms (both lesional and functional), the percentage of which has mostly increased during the last 15 years, are probably a consequence of 'suffering' during either the foetal or neo-natal stage. It could be that this 'suffering' gives rise to a minute isolated lesion within the sub-cortical motor centres (microlesions as described by neuropaediatricians). In situation, there are intermediary forms and come between the organic oculomotor disturbances of the cerebral palsied child and the strictly functional strabismus of the child who is otherwise normal. We have modified and adapted our diagnoses and techniques in view of the multiplicity of such forms and their resistance to conventional therapy. A better understanding of the process responsible, use of more sensitive methods of exploration, a longer period of functional readaptation and new techniques (particularly surgery-) all have a part to play in the new therapy. Where operating is concerned changes apply to the time selected, technique and number of operations performed: two or three stages are necessary. We give a general view of the extreme variety of clinical forms of strabismus and their causes and then give diagnoses and outlines for therapy based on four different type-cases. Clinical facts indicate that in severe forms it is possible to obtained a cure (bifoveality) on condition that the sub-cortical organic damage is very slight and that the optomotor dysfunction (which is its consequence) can be inhibited through satisfactory cortical functioning. However, such treatment requires rigourous participation of both the family and the child over a period of years. Unfortunately this treatment is possible only in a small percentage of cases.
棱镜在斜视治疗中的应用不仅因作者不同而各异,而且同一作者也常常会随着时间的推移改变其对该问题的看法。在前一种情况下,病因病理概念和治疗目的各不相同;在后一种情况下,理论知识的改进以及检查技术和治疗等方面的新可能性发挥了作用。出于后一个原因,我们希望提供补充信息,并总体修订1973年发表的关于棱镜应用的出版物(18)。修订版绝不会使先前的工作无效,它使其与时俱进,并提供了关于严重斜视形式的更多信息,这些严重斜视在首次出版时构成了失败组。这些严重形式(包括损伤性和功能性)在过去15年中大多有所增加,可能是胎儿期或新生儿期“患病”的结果。可能这种“患病”会在皮质下运动中枢产生微小的孤立病变(神经儿科医生所描述的微病变)。实际上,存在中间形式,介于脑瘫儿童的器质性眼球运动障碍和其他方面正常儿童的严格功能性斜视之间。鉴于此类形式的多样性及其对传统疗法的抵抗性,我们已修改并调整了我们的诊断和技术。对病因过程的更好理解、使用更敏感的检查方法、更长时间的功能重新适应以及新技术(特别是手术)都在新疗法中发挥作用。在手术方面,改变适用于所选时间、技术和手术次数:需要分两到三个阶段进行。我们概述了斜视极其多样的临床形式及其病因,然后基于四种不同类型的病例给出诊断和治疗概要。临床事实表明,在严重形式中,如果皮质下器质性损伤非常轻微,并且视动功能障碍(作为其后果)可以通过令人满意的皮质功能得到抑制,就有可能实现治愈(双眼单视)。然而,这种治疗需要家庭和孩子在数年时间里严格参与。不幸的是,这种治疗仅在一小部分病例中可行。