Pigassou-Albouy R
J Fr Ophtalmol. 1980 Feb 20;3(1):45-56.
The mechanism of inhibition of binocular vision (at a cortical level in the Pavlovian sense) in convergent strabismus is described. The study concerns both those patients whith no amblyopia and those whose amblyopia has been cured. Part one of the article describe the clinical characteristics of this cortical inhibition as shown by the polarised light stereoscopic projector. Some basic concepts with this therapeutic application are described in part two. In this paper the author suggests a relationships between the severity of inhibition of binocularity and the degree of disturbance in spatial localisation. The greater the inhibition at a cortical level, the more disturbed is the localisation of objects in space in binocular vision. The treatment of inhibition must include precipitating causes that is to say the disturbed binocular relationships of abnormal retinal correspondence and is complete when cortical inhibition and abnormal retinal correspondence has been completely rectified. On the other hand there are occasions when it is not possible to eliminate abnormal retinal correspondence, this is especially true in older patients, and under this circumstances, it is better to accept inhibition of binocular function than to have diplopia.
本文描述了共同性斜视中双眼视觉抑制（从巴甫洛夫意义上的皮质层面）的机制。该研究涉及无弱视患者以及弱视已治愈的患者。文章第一部分描述了偏振光立体投影仪所显示的这种皮质抑制的临床特征。第二部分阐述了这种治疗应用的一些基本概念。在本文中，作者提出了双眼性抑制的严重程度与空间定位障碍程度之间的关系。皮质层面的抑制越强，双眼视觉中物体在空间中的定位就越受干扰。抑制的治疗必须包括引发原因，也就是说异常视网膜对应导致的双眼关系紊乱，当皮质抑制和异常视网膜对应完全纠正时治疗才完成。另一方面，有时无法消除异常视网膜对应，在老年患者中尤其如此，在这种情况下，接受双眼功能抑制比出现复视要好。
